No, artificial respiration would not save a person who has cyanide poisoning. Cyanide inhibits cellular respiration by blocking the enzyme cytochrome c oxidase in the mitochondria, preventing the cells from using oxygen. Therefore, providing artificial respiration would not be effective in delivering oxygen to the cells and reversing the effects of cyanide poisoning. Immediate medical intervention with antidotes such as hydroxocobalamin or sodium thiosulfate is crucial in treating cyanide poisoning.
A person can get the supply of oxygen for artificial respiration from the cylinders attached to the machines.
an PERSON GETS ARTIFICIAL OXYGEN FROM THE CYLINDER that are attached to the hospitals.
Artificial Respiration is a device or machine that allows a patient or person needing assistance for breathing Breath. This machine acts as the lungs of the person where oxygen can go in and out sustaining life in the body.
yes, the person can be kept in artificial respiration temporarily or permanently. 1:in a hospital a person is given artificial supply of oxygen ,when he is in coma,as long as he can survive under such condition . 2:and for example when a person drowns ,he or she is given artificial respiration.
blood flow to the heart
Yes, the person can be kept in artificialrespiration temporarily or permanently. 1:in a hospital a person is given artificial supply of oxygen ,when he is in coma, as long as he can survive under such condition . 2:and for example when a person drowns ,he or she is given artificialrespiration.
When giving artificial respiration, you are helping to provide the person with the oxygen they need to breathe, but you are not giving them carbon dioxide. The person exhales carbon dioxide naturally as part of the respiration process, and providing artificial respiration helps maintain their oxygen levels while allowing them to get rid of excess carbon dioxide.
Cyanide poisoning occurs because cyanide inhibits the enzyme cytochrome c oxidase, which is one of the enzymes that enable cells to use oxygen. Antidotes for cyanide poisoning tend to focus on binding the cyanide ion so that this inhibition stops. The liver is capable of metabolizing cyanide as well, to take care of any small residual amounts remaining.There are several ways to accomplish this. Nitrites and/or 4-dimethylaminophenol both convert hemoglobin to methemoglobin, which binds tightly with cyanide. This causes problems of its own, though ... methemoglobin cannot carry oxygen, and it's necessary to convert a lot of hemoglobin to methemoglobin to be sure there's an excess to bind all the cyanide, which leaves the person treated with methemoglobinemia ... which then must be treated separately (usually by treatment with methylene blue). Thiosulfates react with cyanide to convert it to thiocyanide, which is much less toxic. However, the reaction does not occur quickly in the body, so this treatment must be supplemented with one of the others (usually nitrates/4-DMAP). Cyanide is present in one form of vitamin B12, so treatment with the cyanideless form hydroxocobalamin will bind cyanide in the harmless cyanocobalamin. Cobalt ions in general will bind cyanide, but cyanocobalt complexes are themselves generally toxic, so this is not necessarily much of an improvement. It is fast, though, and is sometimes used (in the form of the dicobalt EDTA complex) in cases of severe cyanide poisoning. Administration with glucose helps mitigate the toxic effects of the cyanocobalt complexes.
A person can obtain a supply of oxygen for artificial respiration from oxygen tanks, oxygen concentrators, or portable oxygen cylinders. These can be provided by medical facilities, emergency services, or home healthcare providers.
You watch for a rise in the person's chest while blowing air into their mouth.
Cyanide poisoning occurs because cyanide inhibits the enzyme cytochrome c oxidase, which is one of the enzymes that enable cells to use oxygen. Antidotes for cyanide poisoning tend to focus on binding the cyanide ion so that this inhibition stops. The liver is capable of metabolizing cyanide as well, to take care of any small residual amounts remaining.There are several ways to accomplish this. Nitrites and/or 4-dimethylaminophenol both convert hemoglobin to methemoglobin, which binds tightly with cyanide. This causes problems of its own, though ... methemoglobin cannot carry oxygen, and it's necessary to convert a lot of hemoglobin to methemoglobin to be sure there's an excess to bind all the cyanide, which leaves the person treated with methemoglobinemia ... which then must be treated separately (usually by treatment with methylene blue). Thiosulfates react with cyanide to convert it to thiocyanide, which is much less toxic. However, the reaction does not occur quickly in the body, so this treatment must be supplemented with one of the others (usually nitrates/4-DMAP). Cyanide is present in one form of vitamin B12, so treatment with the cyanideless form hydroxocobalamin will bind cyanide in the harmless cyanocobalamin. Cobalt ions in general will bind cyanide, but cyanocobalt complexes are themselves generally toxic, so this is not necessarily much of an improvement. It is fast, though, and is sometimes used (in the form of the dicobalt EDTA complex) in cases of severe cyanide poisoning. Administration with glucose helps mitigate the toxic effects of the cyanocobalt complexes.
The two types of artificial respiration are mouth-to-mouth resuscitation, which involves giving breaths directly into a person's mouth, and chest compressions with rescue breathing, which involves combining chest compressions (CPR) with rescue breaths to help a person breathe and circulate oxygenated blood.