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Complete the flowchart describing the interaction of cortisol with intracellular receptors?
If you're using MasteringBiology on this then look at the third hint and you'll be able to figure it out.
Seriously, look at the third hint if you can to better understand it. It includes pictures.
If you can't or are just too lazy to, here's what I got:
1) Cortisol passes through the plasma membrane into the cytoplasm.
2) Cortisol-receptor complex forms in the cytoplasm.
3) Cortisol-receptor complex enters the nucleus where it binds to genes.
4) Cortisol-receptor complex acts as a transcription factor.
5) The transcribed mRNA is translated into a specific protein.
What else can I help you with?
What do intracellular receptors do?
The intracellular receptors differ from membrane receptors based on their location. Membrane receptors are usually on the plasma membrane but the intracellular receptors are found inside the cell.
What type of receptors is within a cell?
Intracellular receptors. They are receptor proteins found on the inside of the cell, typically in the cytoplasm or nucleus
Which hormone has intracellular receptors?
Steroid hormones, including estrogen, testosterone, and cortisol, have intracellular receptors. These hormones can easily pass through the cell membrane and bind to intracellular receptors located in the cytoplasm or nucleus, where they exert their effects on gene expression.
Hydrophobic signal molecules can interact with what intracellular receptors?
Hydrophobic signal molecules can interact with intracellular receptors, such as nuclear receptors and cytoplasmic receptors. These receptors are typically located inside the cell and can directly bind the hydrophobic signal molecules, leading to changes in gene expression and cellular function.
What keeps intracellular receptors from binding to DNA?
chaperone proteins (chaperonins)
The molecules that convert extracellular signals into intracellular ones are?
Cell surface receptors, such as G-protein coupled receptors, receptor tyrosine kinases, and ligand-gated ion channels, are responsible for converting extracellular signals into intracellular ones. These receptors can initiate various downstream signaling pathways that ultimately regulate cellular responses.
How do steroid hormones bind to intracellular receptors?
Steroid hormones bind to intracellular receptors by passing through the cell membrane and attaching to the receptor inside the cell. This binding activates the receptor, allowing it to move into the cell's nucleus and regulate gene expression.
What keeps intracellular receptors from binding to DNA before a hormone binds to the receptor?
chaperone proteins (chaperonins)
What is an example of a signal molecule that can bind to an intracellular receptor and thereby cause a gene to be turned on or off?
One example is the steroid hormone cortisol. Cortisol can diffuse across the cell membrane and bind to intracellular receptors called glucocorticoid receptors. This complex can then enter the nucleus and regulate the expression of specific genes, turning them on or off.
Why does atropine effect histamine receptors?
It doesn't atropine only acts on muscarinic receptors (it's a competitive antagonist here for ACh), but histamine acts on different receptors (histamine receptors). There is no direct interaction between atropine and histamine receptors
Is estrogen able to get through the cell membrane?
Yes, estrogen can pass through the cell membrane because it is a lipophilic (fat-soluble) molecule. It easily diffuses through the lipid bilayer of the membrane and binds to intracellular estrogen receptors. This interaction then triggers various cellular responses, influencing gene expression and cellular functions.
What are ionotrophic and metabotroph receptors?
Ionotropic receptors are a type of neurotransmitter receptor that directly gates ion channels when activated, leading to rapid changes in membrane potential. Metabotropic receptors, on the other hand, are G protein-coupled receptors that activate intracellular signaling cascades upon neurotransmitter binding, resulting in slower and longer-lasting cellular responses.
