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Blood and urine tests done at the onset of an attack show whether there are elevated or depressed levels of potassium.

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What is hypokalemic periodic paralysis?

The level of potassium in the blood falls in the early stages of a paralytic attack.


What is hyperkalemic periodic paralysis?

The level of potassium in the blood rises slightly or is normal.


What is the difference between hypokalemic and hyperkalemic periodic paralysis?

Hypokalemic periodic paralysis is characterized by low levels of potassium in the blood during attacks, leading to muscle weakness or paralysis. In contrast, hyperkalemic periodic paralysis is characterized by high levels of potassium during episodes, resulting in muscle stiffness or weakness. Both conditions are genetic and involve dysfunction in ion channels that regulate potassium levels in muscle cells.


In familial periodic paralysis how does the paralysis disappear quickly when potassium ions are administered IV?

Periodic paralysis disorders are rare genetic mutation which causes a malfunction in certain ion channels in the muscle membrane that affect muscle strength. There are two major forms, hypokalemic and hyperkalemic, each caused by defects in different genes. Hypokalemic periodic paralysis causes attacks of muscle weakness or paralysis when the level of potassium in the blood drops, while in hyperkalemic, it rises slightly or is normal. During attacks of muscle weakness, potassium moves from the blood into muscle cells. This makes the cell unable to contract properly. Rapidly absorbed boluses of liquid potassium are generally needed to abort an attack and IV potassium is seldom justified unless the patient is unable to swallow. It works more rapidly given IV rather than ingested orally because the K+ions are inserted right into the blood stream rather than being digested in the digestive system and taking it's time to reach the blood stream.


How is periodic paralysis diagnosed?

A complete medical history, physical, neurological exam, electrical tests, blood and urine tests, challenge tests and genetic tests.


What causes paralysis attack?

A paralysis attack, often referred to as a stroke or transient ischemic attack (TIA), can result from a disruption in blood flow to the brain, leading to brain cell damage. This disruption can be caused by a blood clot (ischemic stroke), a burst blood vessel (hemorrhagic stroke), or severe narrowing of the arteries. Other potential causes include trauma, certain medical conditions, and neurological disorders. Risk factors include high blood pressure, diabetes, smoking, and high cholesterol levels.


What is the chemical composition of Zocor?

Simvastatin is the chemical composition the above drug. It is used to keep the blood thin. So that it should not clot in the blood vessel of heart or brain. Which causes heart attack or stroke or paralysis.


Can blood pressure cause a paralysis?

yes it can. If high blood pressure is left untreated, it can cause paralysis, loss of bodily function and motor skills are the result.


Why do physicians monitor a patient's enzyme levels if a heart attack has occurred?

Physicians monitor a patient's enzyme levels if they suspect that the patient is having a heart attack because changes happen over time in blood levels of heart enzymes indicate the heart muscle's health.


What are complications of paralysis?

Complications of paralysis can include muscle atrophy, pressure sores, urinary tract infections, blood clots, and respiratory issues. It is important for individuals with paralysis to work closely with healthcare professionals to prevent and manage these complications.


What is the method of treatment for hypokalemic periodic paralysis?

Severe respiratory weakness from hypokalemic PP may require intensive care to ensure adequate ventilation. Potassium chloride may be given by mouth or intravenously to normalize blood levels.


How decreased blood supply effect muscle activity?

Decreased blood supply in the muscle can cause anoxia then paralysis.