Calcitonin is the hormone that responds to high levels of calcium in the blood by promoting calcium deposition in the bones, thus helping to lower blood calcium levels.
The answer to this is the Parathyroid. The Parathyroid acts to maintain calcium levels in the blood. Thus, if there is a irregularity in the calcium levels, there is also most likely an irregularity in the Parathyroid.
The parathyroid glands are responsible for monitoring the calcium level in the blood. When the calcium level is too low, the parathyroids produce parathyroid hormone (PTH) to increase the calcium level in our blood.
Parathyroid hormone (PTH) and calcitonin are the main hormones that regulate blood calcium levels. PTH increases blood calcium levels by promoting calcium release from bones and enhancing absorption from the intestines, while calcitonin works to lower blood calcium levels by aiding calcium deposition in bones.
After a parathyroidectomy, the calcium levels in the blood are monitored through regular blood tests. The healthcare provider will check the blood calcium levels and adjust calcium supplements or medications as needed to maintain normal levels. Symptom monitoring may also be used, watching for signs of hypocalcemia or hypercalcemia.
Bones have got very large reserve of calcium. So under the influence of calcitonin hormone and parathyroid hormone, the blood calcium level is maintained in very narrow range. Vitamin D is important for absorption of the calcium and also plays some role in maintaining the blood calcium level.
Osteoclastic activity increases the blood calcium level. Osteoblastic activity nullify the effect of osteoclastic activity. Both go hand in hand to rearrange the orientation of calcium lines in the bones, as per requirement of the stresses on the bones.
When blood calcium levels keep increasing, it can lead to hypercalcemia. This can cause symptoms such as fatigue, constipation, confusion, and in severe cases can result in kidney stones, bone loss, and heart problems. It's important to identify and address the underlying cause of the elevated calcium levels.
The hormone produced by the para follicular cells of the thyroid gland is calcitonin (CT). CT can decrease the level of calcium in the blood by inhibiting the action of osteoclasts, the cells that break down bone extracellular matrix. The secretion of CT is controlled by a negative feedback system. When its blood level is high,calcitonin lowers the amount of blood calcium and phosphates by inhibiting bone resorption (breakdown of bone extracellular matrix) by osteoclasts and by accelerating uptake of calcium and phosphates into bone extracellular matrix.
The parathyroid glands play a crucial role in regulating calcium ion levels in the blood. They secrete parathyroid hormone (PTH), which increases blood calcium levels by promoting the release of calcium from bones, reabsorption in the kidneys, and absorption from the intestines. Additionally, the thyroid gland influences calcium regulation through the secretion of calcitonin, which lowers blood calcium levels by inhibiting bone resorption. Together, these glands maintain calcium homeostasis essential for bone health.
The hormone Calcitonin has the effect of reducing blood calcium levels. The hormone comes from the thyroid gland and works in basically three ways. 1. It works to slow absorption of calcium by the intestine. 2. It also inhibits the osteoclast (cells in bone that break down old bone tissue) that raise blood calcium levels. 3. It causes osteoblast (cells in bone that lay down new bone matrix) to form new bone. This explanation is at it's simplest form of course.
This can cause kidney damage due to your body not being able to handle the high levels OF calcium