Renin
Nitric oxide is a vasodilator that can be inactivated by proteolytic enzymes such as superoxide dismutase or hemoglobin. These enzymes can break down nitric oxide, reducing its vasodilatory effects.
No. It is a beta-1 receptor blocker, and an inhibitor of angiotensin converting enzyme (ACE).
Kidneys release the hormones called as erythropoietin. It release the alfacalcidol in your blood. Also called as 1, 25 cholecalciferol. It converts the angiotensin two from angiotensin one and release into the blood. This rise the blood pressure.
The enzyme that converts galactose into glucose 1-phosphate is galactokinase. This enzyme phosphorylates galactose to form galactose 1-phosphate, which can then be converted into glucose 1-phosphate through further metabolic pathways.
Atenolol is a beta blocker and Benazepril is an ACE inhibitor(angiotensin converting enzyme inhibitor) Both can be used for the lowering of blood pressure but they work in different ways. Atenolol blocks the beta-1 receptors in the heart. Beta 1 receptors usually make the heart beat faster and more forcefully. When these receptors are blocked, therefore, it leads to both decreased blood pressure and decreased heart rate. Benazepril blocks the conversion of angiotensin I to angiotensin II. Angiotensin II is an hormone that causes the blood vessels to constrict, or narrow, making the blood pressure increase. Therefore, by blocking the production of this hormone, benazepril causes the blood vessels to do the opposite, and dilate, or open up, causing blood pressure the drop. Atenolol works directly on the heart and therefore affects the heart rate more than Benazepril does.
The enzyme that synthesizes starch from glucose-1-phosphate is starch synthase. This enzyme catalyzes the condensation reaction of glucose molecules to form the starch polymer.
Depends on enzyme purity, activity and incubation conditions.
1. Temperature. 2. P.H level. 3. Enzyme concentration
1 centimeter converts to approximately 0.0328 foot.
1 liter converts to about 2.1 US pints.
if there is any inborn errors with enzyme B means, there will be a malfunctioning... so here the enzyme can not convert comp 1 to comp 2... so automatically enzyme C wont be having any function!!
In step 1 of the lock-and-key model of enzyme function, the substrate (the key) fits into the enzyme's active site (the lock) with precise specificity. This interaction forms an enzyme-substrate complex, allowing the enzyme to stabilize the transition state and facilitate the chemical reaction. The model emphasizes the complementary shapes of the enzyme and substrate, illustrating how enzymes are selective in their action on specific substrates.