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Bacterial resistance to antibiotics mainly arises through genetic mutations and horizontal gene transfer, where bacteria acquire resistance genes from other bacteria. These mechanisms can alter the target sites of antibiotics, enhance efflux pumps that expel the drugs, or produce enzymes that inactivate the antibiotics. Additionally, selective pressure from the overuse and misuse of antibiotics accelerates the survival of resistant strains, leading to the proliferation of resistant bacteria in various environments.

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Why do plasmids contain a gene for antibiotic resistance?

Plasmids contain antibiotic resistance genes because these genes provide a survival advantage to the bacteria in the presence of antibiotics. Bacteria can pick up plasmids with antibiotic resistance genes through horizontal gene transfer, allowing them to survive in environments with antibiotic exposure. This is a common mechanism for bacteria to acquire resistance traits and poses a challenge for antibiotic treatment.


What is bacteria passing resistance to a medicine on to offspring an example of?

Bacteria passing resistance to a medicine on to offspring is an example of vertical gene transfer, where genetic material is transferred from parent to offspring. This mechanism allows for the inheritance of beneficial traits, helping bacteria survive in the presence of antibiotics.


What does 'mechanisms of microbial drug resistance' mean?

exactly what it says. it is a mechanism that confers drug resistance to microbes. exactly what it says. it is a mechanism that confers drug resistance to microbes.


The resistance of an organism to disease is known as?

The medical term for disease resistance is immunity.


What is the evidence that the MLS resistance in staphylococci originated in Streptomyces erythreus?

Streptomyces erythreus naturally produces macrolide antibiotics. It must have a protection mechanism so its protein does not attack itself. This mechanism is most commonly methylation of the 23S rRNA target, and an efflux pump for macrolides. The mechanism of resistance in MLS Staphylococci is the same as Strep erythreus. It is more likely the bacteria shared a simple gene that codes for these resistance mechanisms rather than staph naturally becoming resistant for no competitive reason.


What is bacteria resistance?

Bacteria resistance refers to the ability of bacteria to withstand the effects of antibiotics or other antimicrobial drugs, reducing or eliminating their effectiveness in killing the bacteria. This resistance can develop through genetic mutations or acquisition of resistance genes from other bacteria, leading to treatment challenges and increased risk of uncontrolled infections.


What sugar does RNA have that is different from the sugar in the morning?

Which mechanism explains the phenomenon


Can people glide?

Without an air resistance mechanism, no.


Why does antibiotic resistance evolve in bacteria?

A random mutation in one bacteria can result in this. think of a huge population of bacteria. billions of bacteria. and only 5 or so have the resistance by a random chance (random mutation). the antiobiotics will kill all of bacteria, except for the 5 with the resistance. Then, only those 5 will reproduce. since they reproduce asexually, this resistance will be passed on to all of the daughter. Then, all of sudden, there are a lot of bacteria around that are resistant to the antibiotic... it can also occur by conjugation, which is when a bacteria inserts its DNA into another bacteria. this can result in the second bacteria having the resistance too. this is a very basic description of the process.


Who benefits from a mutation that causes antibiotic resistance in bacteria?

The bacteria benefits.


How is antibiotic resistance an adaptation?

The trait giving bacteria antibiotic resistance has become common, giving bacteria with the trait a selective advantage.


When was Mutations of Bacteria from Virus Sensitivity to Virus Resistance created?

Mutations of Bacteria from Virus Sensitivity to Virus Resistance was created in 1943.