To diagnose an inferior myocardial infarction (MI) using an ECG, the key criteria include the presence of ST-segment elevation in leads II, III, and aVF, indicating inferior wall involvement. Additionally, reciprocal changes may be observed in the lateral leads (I and aVL) as ST-segment depression. The identification of Q waves in these inferior leads can also support the diagnosis of an inferior MI, especially if present in conjunction with the ST-segment changes.
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recent MI is after the 24hrs from the symptoms onset with Q wave on the ECG
CKMB and Troponin marker tests are used for diagnosis of a myocardial infarction. (heart attack). Keeping in mind that a positive blood marker is but one of three criteria used. For a diagnosis of an MI two of three criteria must be met. 1. positive elevation of the st segment in an ECG, 2. Symptoms consistent with myocardial infarction and/or 3. a positive troponin or CKMB. CKMB measures creatine kinase. This is a by product in blood from muscle damage. As the heart is a muscle this can be indicative of cardiac damage. This test is less reliable than a troponin test as results can be altered by other muscle damage. CKMB is not cardiac specific. Troponin is now the most commonly used test for cardiac damage. It is cardiac specific.
An anterior myocardial infarction is generally considered more severe than an inferior myocardial infarction. This is because an anterior MI affects a larger portion of the heart, including the left ventricle, which is responsible for pumping oxygenated blood throughout the body. Inferior MIs typically involve a smaller portion of the heart and may have a better prognosis.
A significant portion of Acute Coronary Syndrome patients have few ECG changes. They are diagnosed based largely on positive enzymes and those aggressively treated early have much better outcomes.
Signs and symptoms include, Hypotension,clear lung sounds , JVD and pitting edema. Pt's who are burping constantly and have an absent " P " wave on their ECG should also be considered suspect for a right sided MI
An inferior wall mycardial infarction is a heart attack involving the inferior portion of the left ventricle, and in many cases the right ventricle. This is caused by occlusion of the right coronary artery. This can also indicate ischemia (poor oxygenation) of the AV node and bundle of HIS, as these structures are also supplied by the right coronary. An inferior wall MI can be seen in the inferior leads II, III, and AVF.
A patient with an inferior wall myocardial infarction (MI) is at risk for several complications, including arrhythmias, particularly bradycardia and atrioventricular (AV) block, due to potential involvement of the right coronary artery and its branches. Additionally, these patients may experience heart failure or cardiogenic shock, especially if there is significant myocardial damage or if the MI is extensive. There is also a risk of recurrent ischemic events and complications related to heart function. Monitoring and prompt intervention are crucial in managing these risks.
You need to talk to the people at Baker College. They will have the criteria necessary for the transfer of credits.
Heart block or bradycardia is more likely after an inferior myocardial infarction (MI) because the right coronary artery, which supplies blood to the inferior part of the heart, can affect the conduction pathways, particularly the atrioventricular (AV) node. Damage to the AV node can lead to a disruption in the electrical signals that regulate heartbeats, resulting in slower heart rates (bradycardia) or heart block. Additionally, inferior MIs can cause increased vagal tone, further contributing to bradycardia.
Usually ST segment elevation... but it doesn HAVE to occur... many people have non st segment MI's (myocardial infarctions) where the only indicator is abnormal blood labs like high CPK or troponins