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Here are the facts as they are known: Elvis Aaron Presley died at the age of 42 on 16 August 1977. He was found on the floor of his bedroom's ensuite (bathroom), and he was rushed to the Baptist Memorial Hospital in Memphis. However, he was pronounced dead on arrival. His post mortem stated that he had died of cardiac arrhythmia - a form of heart attack. His autopsy results will not be made public until 50 years after his death - in 2027. Until that time any guesses as to the manner of his untimely passing will be just that.
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absence of marker material when the lung perfusion scan for the area is normal suggests lung disease
A normal result of an exercise stress test shows normal electrocardiogram tracings and heart rate, blood pressure within the normal range, and no angina, unusual dizziness, or shortness of breath.
Angina pectoris, commonly referred to as just "angina", is chest pain caused by ischemia, or shortage of oxygenated blood supply, to the heart muscle. Unlike a heart attack, angina does not result in permanent damage to the heart muscle.
a stab wound to the heart can result in cardiac tamponade
cardiac dysrhythmias
angina
Cardiac ischemia occurs as a result of insufficient blood flow to the coronary blood vessels. This often occurs due the formation of plagues in the coronary arteries. These plagues narrow the interior of these blood flows resulting in less blood flow to the heart. Angina (or chest pain) sometimes occur as a symptom of cardiac ischemia. The heart has the highest oxygen consumption per tissue mass when compared to other organs in the human body. Blood will flow to the heart during its diastole phase. The heart's vasomotor tone is often controlled by its endothelium where healthy endothelial cells will release nitric oxide to cause vasodilation to improve blood flow. However, endothelial cells which are damaged by plague cannot produce nitric oxide as efficiently so the presence of plagues will decrease vasodilation and this will hinder blood flow. Angina will occur when the oxygen supply does not meet the heart's demand. Propanolol can be used to treat cardiac ischemia. It is a non-selective beta-blocker. It can limit the heart rate by limiting the release to catecolamines. The diastolic coronary perfusion time will hence increase and this is favorable for cardiac ischemia. It also decreases due excitability of the heart and hence reduces the myrocardial oxygen demand.
loss of oxygen.... the mitochondria in the cardiac muscle need it to continue to produce energy to keep the muscles contractiong without it the muscles seize from lactic acidosis a byproduct anaerobic respiration.
This is a phrase you probably found in a doctor's chart or pathology report somewhere. The term "secondary" means "as a result of some other process or finding". The phrase "variant of" means "an unusual but not abnormal or disease-causing condition". "Normal hepatic perfusion" is the typical pathway which blood vessels bring blood into and out of the liver. Putting all the pieces together, "most likely secondary to a variant of normal hepatic perfusion" means this finding was probably caused by a slightly different way that the blood vessels in the liver hooked up and branched out. You would have to consult with your doctor to be sure, but this doesn't sound like a severe problem at this time.
A reduced amount of blood is pumped to the body by the heart, the lower chambers of the ventricles are filled with a less than normal amount of blood, and higher than normal blood pressures occur inside the heart?
Cardiac output (CO) is determined by the heart rate (HR) and the volume of blood pumped by each beat (stroke volume - SV). Mathematically, cardiac output can be represented by the equation: CO = HR x SV As such, if total cardiac output falls as a result of decreased stroke volume, the heart rate can increase to keep the total cardiac output normal, to a certain extent. Stroke volume is more complicated; it is determined by many different factors, including preload, afterload, competence of the atrioventricular valves, ventricular cavity size, and the strength of the squeeze of the cardiac muscle, amongst others. Any change in one of these factors requires a compensation in one or more of the others to maintain cardiac output.
Potassium (K+)