answersLogoWhite

0

IP3. Inositol triphosphate. Cut from the membrane by something I can not remember ( a G protein action ) and then this lipid as part of this signal trasduction pathway docks on the ER and Ca(2+) is released to preform the cellular response.

User Avatar

Wiki User

13y ago

What else can I help you with?

Related Questions

Why is the endoplasmic reticulum rough?

Because it is studded with ribosomes that cause it to be rough.


Which cell structures cause the outer surface of the rough endoplasmic reticulum to appear rough?

The ribosomes attached to the outer surface of the rough endoplasmic reticulum are responsible for its rough appearance. These ribosomes are responsible for protein synthesis, and their presence gives the rough endoplasmic reticulum its distinctive texture.


What would be the effects of adding the sequence KDEL to insulin?

KDEL an amino acid sequence which signals that a protein belongs in the endoplasmic reticulum. Attaching it to insulin would cause the insulin to be retained within the endoplasmic reticulum.


What does sarcoplasmic reticulum do?

The Sarcoplasmic Retiulum releases calcium ions that will cause troponin/tropomyosin complex to move. This exposes the binding sites on actin and allows the cross-bridges of myosin to bind to the actin binding sites.


If Calcium concentrations were low muscle contractions might fail because?

It is the release of calcium from the sarcoplasmic reticulum that then binds with actin-myosin ATPase to cause contractions. Low calcium would first affect the release of acetylcholine from the pre-synaptic terminus.


What do impulses that move down the T tubules most directly cause?

Impulses that move down the T tubules most directly cause the release of calcium ions from the sarcoplasmic reticulum in muscle cells. This calcium release triggers muscle contraction by interacting with the contractile proteins within the muscle fibers.


What would happen if the endoplasmic reticulum stopped working?

Lipid and steriod synthesis will not work properly, toxins will not be detoxified properly, and there may be problems with intracellular calcium signaling, which can be very disruptive to cellular process as calcium is a very import signaling molecule in cells.


Why don't prokaryotes have endoplasmic reticulum?

they don't have organelles, since the cytoplasm does the metabolic work, and technically we will only find the circular DNA in the nucleoid region and some ribosomes (which are NOT organelles, 'cause an organelle is a cellular structure that is protected by a membrane, like mitochondria, chloroplasts) in a prokaryotic cytoplasm.


Normally stored in the terminal cisternae of the sarcoplasmic reticulum?

Calcium ions are normally stored in the terminal cisternae of the sarcoplasmic reticulum in muscle cells. When muscle contraction is initiated, these stored calcium ions are released into the cytoplasm, triggering muscle contraction.


Why is cox enzyme attached to inner surface of endoplasmic recticulum?

Cox enzyme is attached to the inner surface of the endoplasmic reticulum because this is where it functions in the process of cellular respiration to generate energy in the form of ATP. This location allows for efficient communication between Cox enzyme and other proteins involved in the respiratory chain within the endoplasmic reticulum membrane.


When is calcium pumped into the sarcoplasmic reticulum?

When Ach enters the synaptic cleft (gap) it open chemically gated sodium channels that starts an action potential spreading through the sarcolemma of the myofibril. This action potential spreads down the T-tubules and "shocks" the sarcoplasmic reticulum into releasing calcium ions.


What period is characterized by calcium ions release into cytosol and depolarization of the sarcolemma and T tubules?

The period that would be characterized by those things would be muscle contraction. Calcium ions are the neurotransmitters that cause contraction. Calcium Ions depolarize the cell and are spread through out the muscle via the T tubules.