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ther are different types of pathogens like bacteria and viruses which cause disease by self replicating.if we talk about bacteria it divide into two with in specific time i.e e.coli divide into 2 after every 20 minutes by replication.it divide its genome into 2 and synthesize new strands according to the semi conservative model.

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What are the three specific steps for destroying pathogens?

Disrupting the pathogen's cell membrane or wall using physical or chemical means. Inhibiting the pathogen's metabolic processes or replication through heat, radiation, or disinfectants. Blocking the pathogen's ability to infect host cells or evade the immune system through antibodies or antiviral drugs.


How does DNA replication compare in eukaryotes and prokaryotes?

DNA replication in prokaryotes occurs in the cytoplasm and typically begins at a single origin of replication, resulting in the simultaneous replication of both strands in a bidirectional manner. In contrast, eukaryotes replicate their DNA in the nucleus, utilizing multiple origins of replication along linear chromosomes, which allows for faster replication of larger genomes. Additionally, eukaryotic replication involves more complex machinery and regulatory mechanisms, including histone modifications and telomere maintenance, compared to the simpler process in prokaryotes. Overall, while the fundamental process of DNA replication is similar, the organizational and regulatory differences reflect the complexity of eukaryotic cells.


What is the importance of DNA replication in the cell reproduction?

DNA replication is significant in the reproduction of cells through the mechanisms of mitosis and meiosis. The DNA replicates so that there will be an extra DNA when the cell multiplies, allowing the "daughters" the chance to reproduce. DNA replication is significant in the reproduction of cells through the mechanisms of mitosis and meiosis. The DNA replicates so that there will be an extra DNA when the cell multiplies, allowing the "daughters" the chance to reproduce.


What is subtractive genomics?

Subtractive genomic strategy is developed by assuming that the novel targets identified in the pathogen should be essential for the pathogen that is it should be involved in the replication, survival and a important component of various metabolic pathways and mechanisms occurring in the pathogen while at the same time should be absent on the host that is human and should have no homologue in human, so that when a drug or a lead compound is designed considering the potential target it should only be against the mechanism and functionality of the pathogen not the host.


Which helps prevent errors in dan replication?

To prevent errors in DNA replication, several mechanisms are in place, including the proofreading ability of DNA polymerases, which can detect and correct mismatched nucleotides during synthesis. Additionally, various repair pathways, such as mismatch repair and nucleotide excision repair, help identify and fix errors after replication. These processes ensure high fidelity during DNA replication, maintaining genetic stability.

Related Questions

Describe two mechanisms that operate to restore homeostasis after infection by a pathogen?

Gaben at valve


What is colonisaion in infection?

As you probably know, colonization and infection are 2 separate events.Colonization occurs when a pathogen enters a wound then replicates.Once you have colonization/replication of a pathogen in a wound, the wound will (most likely) become infected.


An animal insect or human whose body is capable of sustaining the growth of a pathogen is known as what?

An animal, insect, or human whose body can sustain the growth of a pathogen is known as the pathogen's host.


What are the differences in the functions and mechanisms of action between helicase and topoisomerase in DNA replication and repair processes?

Helicase unwinds the double-stranded DNA during replication by breaking hydrogen bonds between base pairs, while topoisomerase helps relieve the tension and supercoiling that occurs ahead of the replication fork by cutting and rejoining the DNA strands. Both enzymes play crucial roles in DNA replication and repair processes, but they have distinct functions and mechanisms of action.


How is bacterial DNA replication different from eukaryotic DNA replication in terms of process and mechanisms?

Bacterial DNA replication is simpler and faster than eukaryotic DNA replication. Bacteria have a single circular chromosome, while eukaryotes have multiple linear chromosomes. Bacterial replication occurs bidirectionally from a single origin, while eukaryotic replication starts at multiple origins. Bacteria use a DNA polymerase III enzyme for replication, while eukaryotes use multiple DNA polymerases.


What are the key differences in the genetic material replication process that DNA uses, while RNA uses a different mechanism?

DNA replication involves the synthesis of a new DNA strand using the existing DNA as a template, while RNA replication involves the synthesis of RNA using DNA as a template. DNA replication is highly accurate due to proofreading mechanisms, while RNA replication is less accurate. Additionally, DNA replication occurs in the nucleus, while RNA replication can occur in the nucleus or cytoplasm.


What are the three specific steps for destroying pathogens?

Disrupting the pathogen's cell membrane or wall using physical or chemical means. Inhibiting the pathogen's metabolic processes or replication through heat, radiation, or disinfectants. Blocking the pathogen's ability to infect host cells or evade the immune system through antibodies or antiviral drugs.


How does DNA replication compare in eukaryotes and prokaryotes?

DNA replication in prokaryotes occurs in the cytoplasm and typically begins at a single origin of replication, resulting in the simultaneous replication of both strands in a bidirectional manner. In contrast, eukaryotes replicate their DNA in the nucleus, utilizing multiple origins of replication along linear chromosomes, which allows for faster replication of larger genomes. Additionally, eukaryotic replication involves more complex machinery and regulatory mechanisms, including histone modifications and telomere maintenance, compared to the simpler process in prokaryotes. Overall, while the fundamental process of DNA replication is similar, the organizational and regulatory differences reflect the complexity of eukaryotic cells.


What is the importance of DNA replication in the cell reproduction?

DNA replication is significant in the reproduction of cells through the mechanisms of mitosis and meiosis. The DNA replicates so that there will be an extra DNA when the cell multiplies, allowing the "daughters" the chance to reproduce. DNA replication is significant in the reproduction of cells through the mechanisms of mitosis and meiosis. The DNA replicates so that there will be an extra DNA when the cell multiplies, allowing the "daughters" the chance to reproduce.


How does the glandular fever pathogen divide inside the body?

The glandular fever pathogen, Epstein-Barr virus (EBV), infects B-cells in the body and replicates within these host cells. It utilizes the host cell's machinery to multiply and spread to other cells. This replication process contributes to the symptoms associated with glandular fever.


How accurate is the process of DNA replication in ensuring the faithful copying of genetic information?

The process of DNA replication is highly accurate in ensuring the faithful copying of genetic information. The enzymes involved in DNA replication have proofreading mechanisms that help to correct any errors that may occur during the process, resulting in a very low error rate.


What is subtractive genomics?

Subtractive genomic strategy is developed by assuming that the novel targets identified in the pathogen should be essential for the pathogen that is it should be involved in the replication, survival and a important component of various metabolic pathways and mechanisms occurring in the pathogen while at the same time should be absent on the host that is human and should have no homologue in human, so that when a drug or a lead compound is designed considering the potential target it should only be against the mechanism and functionality of the pathogen not the host.