The reabsorption of excess neurotransmitter molecules by a sending neuron is called "reuptake." This process helps regulate neurotransmitter levels in the synaptic cleft, ensuring that signaling between neurons is appropriately balanced. Reuptake is a crucial mechanism for maintaining neurotransmitter homeostasis and can influence mood, behavior, and various neurological functions.
Reabsorption via a process called "secretion".
Glomerular Filtration
Molecules that are similar enough to a neurotransmitter to bind to its receptor sites on a dendrite and mimic its effects are called agonists. These can be naturally occurring substances or synthetic drugs that activate receptor sites, leading to similar physiological responses as the original neurotransmitter. Examples include morphine, which mimics endorphins, and nicotine, which mimics acetylcholine.
They are called as vesicles. They are constantly created. They contents the neurotransmitter. Neurotransmitter is required for transmission of the nerve impulse.
The transport mechanism for a neurotransmitter across the synaptic cleft is called exocytosis. During exocytosis, neurotransmitter-filled vesicles fuse with the presynaptic membrane, releasing the neurotransmitter into the synaptic cleft where it can then bind to receptors on the postsynaptic membrane.
Reuptake
The small space between two neurons where neurotransmitter molecules are released is called the synapse.
Neurotransmitter molecules are removed from a synapse through a process called reuptake or enzymatic degradation. In reuptake, the neurotransmitter is taken back up into the presynaptic neuron. In enzymatic degradation, special enzymes break down the neurotransmitter molecules into inactive byproducts.
Reabsorption via a process called "secretion".
Glomerular Filtration
Molecules that are similar enough to a neurotransmitter to bind to its receptor sites on a dendrite and mimic its effects are called agonists. These can be naturally occurring substances or synthetic drugs that activate receptor sites, leading to similar physiological responses as the original neurotransmitter. Examples include morphine, which mimics endorphins, and nicotine, which mimics acetylcholine.
The process is called neurotransmitter synthesis. It involves the conversion of precursor molecules into neurotransmitters by specific enzymes within neurons. This process is crucial for the production of neurotransmitters that are essential for communication between neurons in the brain.
3 ways neurotransmitters can be removed: 1. Reuptake- reabsorption of the neurotransmitter into the neuron. 2. Enzymatic degradation- destruction of the neurotransmitter with special chemicals called enzymes. 3. Diffusion- The neurotransmitter becoming detached from the receptor and drifting out of the synaptic cleft.
neurotransmitters
The substance that is released at an axonal ending to propagate the nerve impulse to the next nerve or muscle is called
They are called as vesicles. They are constantly created. They contents the neurotransmitter. Neurotransmitter is required for transmission of the nerve impulse.
The transport mechanism for a neurotransmitter across the synaptic cleft is called exocytosis. During exocytosis, neurotransmitter-filled vesicles fuse with the presynaptic membrane, releasing the neurotransmitter into the synaptic cleft where it can then bind to receptors on the postsynaptic membrane.