Eating too many antacids can disrupt the natural acidity of the stomach, leading to decreased gastric enzyme activity. The alkaline environment created by excessive antacid use can impair the function of enzymes like pepsin, which requires an acidic pH to effectively break down proteins. This can hinder digestion and nutrient absorption, potentially leading to gastrointestinal issues and deficiencies over time. Additionally, prolonged use may trigger a rebound effect, causing increased stomach acid production once the antacids are discontinued.
Sympathetic activity inhibits gastric secretion by causing vasoconstriction of blood vessels in the stomach lining. Parasympathetic activity stimulates gastric secretion by increasing blood flow and activating gastric glands, leading to increased production of stomach acid and digestive enzymes.
Gastric acid.
The stomach primarily produces gastric acid, digestive enzymes, and mucus. Gastric acid, primarily hydrochloric acid, helps break down food and provides an acidic environment for enzymes to function. The main digestive enzyme is pepsin, which begins protein digestion. Mucus protects the stomach lining from the corrosive effects of acid and helps lubricate food for easier movement through the digestive tract.
Mucus is a thick, gel-like substance produced by mucous membranes, serving to protect and lubricate surfaces in the body, including the gastrointestinal tract. In the stomach, gastric mucus forms a protective barrier that shields the stomach lining from the corrosive effects of gastric acid and digestive enzymes. If mucus is not secreted by the gastric glands, the stomach lining can become damaged, leading to conditions such as gastritis, ulcers, and increased risk of gastrointestinal bleeding.
The human digestive system has several protective mechanisms, primarily the gastric acid and the mucosal barrier. Gastric acid, produced by the stomach lining, helps to kill harmful pathogens and break down food. Additionally, the mucosal barrier, composed of mucus secreted by epithelial cells, protects the stomach and intestines from digestive enzymes and acidic environments. This combination helps to prevent infections and maintain the integrity of the digestive tract.
Sympathetic activity inhibits gastric secretion by causing vasoconstriction of blood vessels in the stomach lining. Parasympathetic activity stimulates gastric secretion by increasing blood flow and activating gastric glands, leading to increased production of stomach acid and digestive enzymes.
Base salts
Gastric means something relating to the stomach/digestive system.
gastric acid
Pre-gastric fermentation is the process of microbial digestion occurring before the food reaches the hosts digestive epithelium (which secrete acids and enzymes) Post-gastric Fermentation has microbial digestion after hosts digestive epithelium.
Gastric acid.
Antacids got their name from their primary function of counteracting (or neutralizing) stomach acidity. The prefix "anti-" means "against," while "acid" refers to the acidic environment in the stomach. These substances, typically containing alkaline compounds, help relieve heartburn and indigestion by raising the pH of gastric acid. Thus, their name reflects their role in combating acidity in the digestive system.
The stomach contains gastric juices for chemical digestion.
Gastric Juice
Gastric Juice
THE GASTRIC CAECA ARE SMALL SAC-LIKE APPENDAGES THAT NORMALLY CONTAIN GASTRIC JUICES THAT ARE INVOLVED IN THE DIGESTION OF FOOD IN THE MID GUT.
secretion