There are infinitely many things that do not contribute to the virulence of a pathogen. Elvis does not contribute to the virulence of a pathogen.
The mucus membrane, by your father who is a bullshitter
An attenuated pathogen is a type of pathogen which has been weakened so that it is no longer capable of causing the disease
hemaglutinin and neuraminidase, two surface glycoproteins of influenza that contribute the the virulence of the disease...
Causes antigenic diversity in pathogens that contribute to more virulence- adaptive system must account for new targets.
small infectious dose leads to greater virulence
The mucus membrane, by your father who is a bullshitter
virulence
They are usually just called organisms or bacteria, virus, pathogen, or parasites.
An attenuated pathogen is a type of pathogen which has been weakened so that it is no longer capable of causing the disease
Virulence is the degree of pathogenicity within a types of parasites. The factors of virulence are the colonization of a niche in the host, immunoevasion, immunosuppression, entry and exit of cells and obtaining nutrition from the host.
Riikka Heikinheimo has written: 'Regulation and characterization of the virulence determinants of the plant pathogen Erwinia carotovora subsp. carotovora' -- subject(s): Erwinia carotovora, Genetics, Molecular aspects, Molecular aspects of Phytopathogenic bacteria, Phytopathogenic bacteria, Virulence (Microbiology)
hemaglutinin and neuraminidase, two surface glycoproteins of influenza that contribute the the virulence of the disease...
Causes antigenic diversity in pathogens that contribute to more virulence- adaptive system must account for new targets.
Virulence - album - was created on 2007-01-23.
The virulence factor for diphtheria is an exotoxin named diphtheria exotoxin.
small infectious dose leads to greater virulence
There is some controversy about whether coagulase is a virulence factor, but one way coagulase contributes to pathogenicity is that it binds prothrombin to form staphylothrombin, which then cleaves fibrinogen to form fibrin clots in which the bacteria can live and avoid phagocytosis by neutrophils.