The Na+/K+ ATPase pumps in the renal tubules and intestines.
aldosterone, antidiuretic hormone (ADH, or vasopressin), atrial natriuretic peptide (ANP), and other hormones.The three hormones responsible for regulating renal sodium and chloride ion reabsorption are;1. Aldosterone2. Atrial natriuretic peptide or ANP3. Angiotensin II
aldosterone
Aldosterone helps the body cope with ongoing stress by increasing sodium reabsorption in the kidneys, which leads to water retention and increased blood volume. This helps to maintain blood pressure during stressful situations.
The Na/K ATP pump, insulin, catecolamins, the kidney (pH balance) and aldosterone.
Short answer: In the kidney, aldosterone increases sodium retention, which increases potassium excretion, which increases H+ excretion, so aldosterone will tend to raise blood pH.Explanation:Aldosterone directly increases sodium retention in the kidney. ?Since all kidney cells have basolaterally located Na+/K+ ATPases, this will cause these exchangers to go into overdrive and dump more K+ into the cells. ?For those cells that have K+ channels, this will increase potassium excretion.Finally, certain cells in your collecting ducts have H+/K+ exchangers (either the alpha or beta intercalated cells). ?So if you have more K+ in the lumen, this will cause these exchangers to kick into overdrive and dump H+ into the lumen, which finally raises blood pH.
Increased Na+ reabsorption
It work on kidneys.It stimulates reabsorption of Na+.
Aldosterone is the hormone that regulates sodium reabsorption in the distal convoluted tubule of the nephron. It acts on the epithelial cells of the tubule to increase the reabsorption of sodium and water, helping to maintain electrolyte balance and blood pressure in the body.
When aldosterone is absent, Na+ reabsorption in the nephron is greatly reduced, and as 30-40g of sodium can be lost in the urine daily. The answer is decreased. Site: Anatomy and Physiology Eighth Edition by Seeley Stephens Tate
aldosterone, antidiuretic hormone (ADH, or vasopressin), atrial natriuretic peptide (ANP), and other hormones.The three hormones responsible for regulating renal sodium and chloride ion reabsorption are;1. Aldosterone2. Atrial natriuretic peptide or ANP3. Angiotensin II
Aldosterone is a hormone responsible for the reabsorption of Na+ and the secretion of K+. It is produced by the adrenal glands that sit atop the kidney (renal).
potassium-sparing diuretics
Aldosterone will cause Na+ to be absorbed from a filtrate
Aldosterone is produced by the adrenal cortex, specifically the outer adrenal cortex called the Glomuerulosa. The adrenal cortex is also composed of an Mid section called the fasciculate which produces cortisone and the inner portion called reticularis which produces adrogen hormones.
The major target of aldosterone is the distal tubule of the kidney, where it stimulates exchange of sodium and potassium. Three primary physiologic effects of aldosterone result:Increased resorption of sodium: sodium loss in urine is decreased under aldosterone stimulation.Increased resorption of water, with consequent expansion of extracellular fluid volume. This is an osmotic effect directly related to increased resorption of sodium.Increased renal excretion of potassium.Knowing these effects should quickly suggest the cellular mechanism of action this hormone. Aldosterone stimulates transcription of the gene encoding the sodium-potassium ATPase, leading to increased numbers of "sodium pumps" in the basolateral membranes of tubular epithelial cells. Aldosterone also stimulates expression of a sodium channel which facilitates uptake of sodium from the tubular lumen.
Aldosterone promotes water retention and reduces urine volume
Aldosterone. Aldosterone acts on the Na+/K+ ATPase pump in the Distal Proximal Tube/Collecting duct in the kidney. Importantly, when this pump action is up-regulated, Na+ re-absorption goes up along with WATER, which is recovered with the sodium ions.