If not enough insulin is produced, the concentration of blood glucose will increase because insulin is responsible for helping glucose enter cells to be used for energy. Without enough insulin, glucose remains in the bloodstream, leading to high blood sugar levels, a condition known as hyperglycemia.
The insulin binds to insulin receptors on the surface of muscle or liver cells. This opens up little holes in the cell membrane called glucose transporters. Glucose flows through the glucose transporter due to the concentration gradient of glucose being higher in the extracellular environment. This is called diffusion. The membrane only stays permeable (open) to glucose so long as there is insulin bound to the receptors on the cell surface. Eventually the insulin is released and the glucose transporter closes. The cell then starts to digest the glucose via complicated processes called glycolysis and oxidative phosphorylation.
bcoz without insulin secreation glucose concentration in the blood is high, and no energy can be store in body, bcoz glucose is our ultimate energy, also without insulin , heart attacks occur and BP high. and beta cell secrete insulin.
insulin resistance. This means that the cells in the body are not responding effectively to insulin, leading to elevated blood sugar levels. Insulin resistance is a key characteristic of Type II diabetes and can contribute to the development of the condition.
The fingertips have a greater concentration of sensory receptors compared to the palms of the hands. This is why our fingertips are more sensitive to touch and able to detect fine details such as texture and temperature with precision.
glucagon.
High insulin levels occur in an attempt to counter high levels of sugar. Type II diabetes is caused by insulin receptors on cells not having much of a response to insulin. Hence, more insulin is released in an attempt to lower the blood sugar levels. This is why high insulin levels are a symptom of Type II diabetes, not a cause.
If not enough insulin is produced, the concentration of blood glucose will increase because insulin is responsible for helping glucose enter cells to be used for energy. Without enough insulin, glucose remains in the bloodstream, leading to high blood sugar levels, a condition known as hyperglycemia.
That would be the peptide hormone called "Glucagon". (GLOO-kuh-gone)
Insulin receptors are located on the surface of cell membranes. They are predominantly found on muscle, fat, and liver cells, where they play a crucial role in regulating glucose uptake and metabolism in response to insulin signaling.
diabetes mellitus
The body doesn't have a fixed number of insulin receptors it "needs," as this can vary based on factors like individual metabolism, insulin sensitivity, and overall health. Insulin receptors are present on nearly all cells, particularly those in muscle, fat, and liver tissues, to facilitate glucose uptake. The number of functional receptors can change in response to factors such as obesity, exercise, and diet. Essentially, the body aims to maintain a balance that allows for effective glucose regulation.
If insulin receptors stopped working, the cells would not be able to take in glucose from the bloodstream effectively. As a result, blood sugar levels would increase because the body would not be able to properly regulate glucose uptake. This could lead to high blood sugar levels (hyperglycemia) and potentially result in symptoms associated with diabetes.
Insulin and epinephrine
A problem with insulin receptors can disrupt homeostasis by impairing the body’s ability to regulate blood glucose levels. When insulin receptors are not functioning properly, cells may not respond effectively to insulin, leading to decreased glucose uptake and increased blood sugar levels. This can result in conditions like insulin resistance or type 2 diabetes, which can further affect metabolic processes and overall bodily function. Ultimately, such disruptions can lead to various health complications if not managed effectively.
The insulin binds to insulin receptors on the surface of muscle or liver cells. This opens up little holes in the cell membrane called glucose transporters. Glucose flows through the glucose transporter due to the concentration gradient of glucose being higher in the extracellular environment. This is called diffusion. The membrane only stays permeable (open) to glucose so long as there is insulin bound to the receptors on the cell surface. Eventually the insulin is released and the glucose transporter closes. The cell then starts to digest the glucose via complicated processes called glycolysis and oxidative phosphorylation.
I think it is the Brain....