When an action potential reaches the end of a neuron, it triggers the release of neuotransmitters such as epinephrine (sympathetic) or achetylcholine (parasympathetic).
Following a threshold potential, voltage-gated sodium channels open, allowing sodium ions to rush into the cell, depolarizing the membrane. This triggers an action potential that spreads down the length of the neuron. Once the action potential reaches the end of the neuron, it triggers the release of neurotransmitters into the synaptic cleft to communicate with the next neuron.
When an action potential reaches the axon terminal of a neuron, it triggers the release of neurotransmitters into the synaptic gap. These neurotransmitters then bind to receptors on the postsynaptic neuron, causing ion channels to open and allow ions to flow in, generating a new action potential in the receiving neuron.
When an action potential reaches the axon terminal of the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic neuron, leading to changes in its membrane potential. This process either excites or inhibits the postsynaptic neuron, depending on the neurotransmitter and receptor type involved.
Disturbances to sensory neurons can cause depolarization of the neuron's membrane, reaching a threshold that triggers an action potential. This action potential then travels along the neuron's axon to the central nervous system, where it is processed and interpreted as a sensory experience.
The presynaptic cell that must have action potentials to produce one or more action potentials in the postsynaptic cell is the neuron releasing neurotransmitters at the synapse. When an action potential reaches the presynaptic terminal, it triggers the release of neurotransmitters into the synaptic cleft, which then bind to receptors on the postsynaptic cell membrane, leading to the generation of an action potential in the postsynaptic cell.
When an action potential reaches the nerve terminal, it triggers the release of neurotransmitters into the synapse.
When an action potential reaches an axon terminal, it triggers the release of neurotransmitters into the synaptic cleft.
When an action potential reaches the nerve terminal, it triggers the release of neurotransmitters into the synapse, which then transmit signals to the next neuron or target cell.
When the action potential reaches the axon terminal, it triggers the release of neurotransmitters into the synapse, which then bind to receptors on the neighboring neuron, continuing the signal transmission.
Following a threshold potential, voltage-gated sodium channels open, allowing sodium ions to rush into the cell, depolarizing the membrane. This triggers an action potential that spreads down the length of the neuron. Once the action potential reaches the end of the neuron, it triggers the release of neurotransmitters into the synaptic cleft to communicate with the next neuron.
When an action potential reaches the end of a neuron's axon, it triggers the release of neurotransmitters from vesicles in the presynaptic terminal into the synaptic cleft. This process is mediated by the influx of calcium ions that enter the neuron during an action potential, causing the vesicles to fuse with the cell membrane and release their contents.
When an action potential reaches the axon terminal of a neuron, it triggers the release of neurotransmitters into the synaptic gap. These neurotransmitters then bind to receptors on the postsynaptic neuron, causing ion channels to open and allow ions to flow in, generating a new action potential in the receiving neuron.
After the action potential reaches the presynaptic terminal, voltage-gated calcium channels open, leading to an influx of calcium ions. This triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic membrane, leading to depolarization and the generation of a new action potential in the postsynaptic neuron.
When an action potential reaches the end of a neuron, it triggers the release of neurotransmitters into the synapse. These neurotransmitters then bind to receptors on the receiving neuron, causing ion channels to open and allowing ions to flow in, which generates a new action potential in the receiving neuron, thus continuing the signal.
depolarization
When the action potential reaches the end of the axon terminals, it triggers the opening of voltage-gated calcium channels. The influx of calcium ions prompts synaptic vesicles filled with neurotransmitters to fuse with the presynaptic membrane. This fusion releases neurotransmitters into the synaptic cleft, where they bind to receptors on the postsynaptic neuron, facilitating communication between neurons.
When an action potential reaches the axon terminal of the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic neuron, leading to changes in its membrane potential. This process either excites or inhibits the postsynaptic neuron, depending on the neurotransmitter and receptor type involved.