The psoas muscle is responsible for stabilizing the spine and allowing it to flex and also allows for rotation of the hips for movement. A lesion in this area would likely cause lower Back pain and would limit a person's range of motion and flexibility. If the lesion were to spread to the right neural foramina, this could cause further complications since this is a nerual pathway down the spinal cord. If the lesion were to spread to this area it could occlude where those nerve fibers exit.
The CNS has INHIBITORY effects on muscles. Loss of this inhibition from an UMN lesion leads to increased muscle activity (spastic paralysis and a + babinski).
if the lesion is in a contractile tissue i.e muscle then active n passive movements are painful and/or restricted in opposite direction of motion.
lactic acid
Motor Point is located where the moter nerve enters the muscle. It is where the muscle is most electically exciteable.
The coronary arteries feed the heart.
Myopathy is a pathologic change of muscle tissues due to disease or injury.myopathy
An UMN lesion may in fact present with atrophy, but it does so over time. With a LMN lesion, the muscle loses its innervation directly and thus will not be receiving any neurological stimulus at all. However, damage to an UMN leaves the corresponding LMN to that particular muscle intact, thus leaving it to fire somewhat irregularly, but firing nonetheless. Macroscopically the muscle is no longer under central control and thus can't perform proper function, but microscopically the muscle fibers are still receiving some neurological input, or signal from the LMN innervating it, thus the muscle integrity appear unaffected at first. Over time, the muscle will atrophy because of a lack of use, but this happens more gradually than with a direct LMN lesion. -Onyx (Medical Student)
paralysis of tongue muscle ipsilateral to lesions (due to damaged hypoglossal nerve)hemiplagia contralateral to lesion (due to damaged corticospinal tract)loss of sensations carried by medial lemniscus - contralateral to lesion
LMNLIt is due to lesion of lower motor neurons i.e. the spinal and cranial motor neurons that directly innervate the muscles.Usually single or individual muscle is affected.Disuse atrophy of muscles.All reflexes are absent.flaccid paralysis occurs.Babinski planter response not elicited.2. UMNLIt is due to upper motor neuron lesion.It involves group of muscles.spastic paralysis occurs.muscle atrophy is not severe.reflexesdeep reflexes are hyperactive.superficial refle;xes;only abdominal,cremastric and anal reflexes are lost.Babinski sign:positive.AI'IGHT,UUUMMM HOPE THIS HELPS :) LOWER MOTOR NEURON LESION: vs UPPER MOTOR NEURON LESION:1)FLACCID MUSCLE SPASTICITY OF MUSCLE2)HYPO-TONIA HYPER-TONIA3)HYPO-REFLEXIA HYPER-REFLEXIA4)PROFOUND MUSCLE ATROPHY MINIMAL MUSCLE ATROPHY5)FASCICULATIONS("TWITCHING") PRESENT FASCICULATIONS ABSNTSO JUST REMEMBER: LOWER = HYPO- EVERYTHING ; UPPER = HYPER- EVERYTHING,WELL, EXCEPT OFCOURSE - THE TWITCHING AND MUSCLE ATROPHY (ITS REVERSED).
The incus is a small muscle in you ear which is connected to the eardrum and malleus. The malleus increases the loudness of sound that enters the ear.
Acetylcholine is the chemical that crosses the synapse causing a muscle to contract. It is released due to an electrical impulse sent from your brain down your nervous system.
Lungs do not contain muscle tissue. air enters your lungs when the muscular diaphragm contracts, pulling your ribs up and out. The diaphragm relaxes when you exhale.