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Why are antibiotics drugs effective against bacteria without hurting humans because the antibiotics rely upon differences in the structure of human and bacteria?
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How do chemists used scientific processes to develops a solution to human bacterial infections?
With Antibiotics
Why do antibiotics usually affect microbial cells and not host cells?
Antibiotics typically target features unique to microbial cells, such as bacterial cell walls, ribosomes, or metabolic pathways that are not present in human cells. For example, many antibiotics inhibit bacterial protein synthesis by binding to ribosomes that differ from those in human cells. Additionally, the structural differences in cell membranes and the presence of specific enzymes allow antibiotics to selectively disrupt microbial functions without harming host cells. This selective targeting minimizes damage to the host while effectively combating bacterial infections.
How do chemists use this fact to create antibiotics that can destroy a bacterial infection without harming human cells?
Antibiotics attack proteins only found in bacteria. Each one targets a specific area, be it their cell wall, cell membrane, protein synthesis centers, and really any area that differs enough from human cells
Why do protein synthesis inhibitor a.k.a antibiotics affect bacteria only but not human cells?
most of the antibiotics kill or inactivate bacteria by inhibitting the protein synthesis... protein synthesis consists of 'transcription' and 'translation'.. the translation process requiers mRNA and ribosomes.Human(eukaryotic) ribosome is different from bacterial(prokaryotic) ribosome... Antibiotics inhibit the protein synthesis by altering the ribosomal constitution.Since human ribosomes are different from bacterial ribosome,the substances which are harmful to bacterial ribosome doesn't harm human ribosomes.. Thus human cells are immune to antibiotics..
Why ionophore antibiotics do not kill even your human cells but only bacterial ones?
Only microbes have ionophones, which are channels in their membranes. By blocking them using antibiotics, the cell will die as they can not bring ions in and out of their cells. Human cells don't have these and so are not affected by those antibiotics.
Explain why antibiotics are effective at killing bacterial cells but do not seem to have an impact on human cells eukaryotic cells Use at least two examples such as tetracycline erythromycin?
Antibiotics target specific structures or processes unique to bacterial cells that are not present in human eukaryotic cells. For example, tetracycline interferes with bacterial protein synthesis by binding to bacterial ribosomes, while erythromycin inhibits the bacterial ribosome's ability to make proteins. Since human cells do not have the same type of ribosomes or protein synthesis mechanisms, antibiotics like tetracycline and erythromycin do not affect human cells the same way they do bacterial cells.
In what structure does Bacterial DNA resemble human DNA?
That they have both DNA Bacterial DNA is a (closed) circle, those of human is lineair (straight) if you stretch it. Bacterial DNA does not have Proteines, Human DNA has Proteines. Bacteria have also RNA The DNA of bacteria is easy to reach , those of human not.
What compound kills bacteria without harming the cells of humans?
Antibiotics such as penicillin and tetracycline are effective in killing bacteria by targeting specific bacterial cell components that are absent in human cells, thus reducing the risk of harm to human cells. However, misuse or overuse of antibiotics can lead to antibiotic resistance in bacteria, diminishing their efficacy over time.
What are the drugs that are used to treat bacterial diseases called?
Most of the drugs that treat bacterial disease are called antibiotics.
Can antibiotics prevent HIV?
because HIV is a virus and antibiotics treat bacterial infections.
How do human diseases cause by bacteria and diseases caused by viruses react to antibiotics?
Viruses ALL ignore antibiotics, Bacterial infections vary in their response depending on the sensitivity of the bacteria to the specific antibiotic.
