No Factor VIII is Antihemophilic factor.
Thrombocytopenia is a low platlet count Thrombocyte = platlet Penia = poorness
factor VIII
Factor VIII is a protein that is typically made in the liver and released into the bloodstream. It is an essential component for normal blood clotting.
In the most common type, Hemophilia A, the person lacks clotting factor VIII. In Hemophilia B, clotting factor IX is lacking.
Haemophilia A - an inhibition of clotting caused by a deficiency in a protein called Clotting Factor VIII Haemophilia B - an inhibition of clotting caused by a deficiency in a protein called Clotting Factor IX Haemophilia C - an inhibition of clotting caused by a deficiency in a protein called Clotting Factor XI -similar to- vonWillebrand's Disorder - an inhibition of clotting caused by a deficiency in a protein called the vonWillebrand's Factor
The main treatment for hemophilia is called replacement therapy. Concentrates of clotting factor VIII or clotting factor IX are slowly dripped or injected into a vein.
Hemophilia A is an X-linked, hereditary bleeding disorder caused by the absence or defect of a blood clotting protein, Factor VIII. As a result, when a person with hemophilia A has a bleeding episode, the bleeding may be prolonged due to the body's inability to form blood clots. Patients who are affected with hemophilia A experience frequent spontaneous bleeding, most commonly into their joints and soft tissues, with bleeding into vital organs that may ... be life-threatening. Bleeding episodes may be painful, and over time, recurrent joint bleeding may result in debilitating destruction of the joints. Currently, patients with hemophilia A are dependent on injections of Factor VIII produced by genetic engineering or purified from human plasma, to help control a bleeding episode. It is estimated that approximately 50,000 individuals worldwide are affected with hemophilia A. Hemophilia A - Clotting Factor VIII Hemophilia B - Clotting Factor IX Hemophilia C - Clotting Factor XI
Hemophilla is a genetic disorder in which there is absence of clotting factor viii, leading to defective clot formation, petechiae bruising and gum bleeding. Treatment is by injecting factor VIII concentrate
There are three types of hemophilia: A, B, CQueen Victoria was a carrier of hemophilia B.Proteins are blood-clotting factors that hemophiliacs lack.Hemophilia A is Clotting Factor VIII deficiency.Hemophilia B is Clotting Factor IX deficiency.Hemophilia C is Clotting Factor XI deficiency.Hemophiliacs do not bleed more than other people, they simply bleed longer.Hemophilia is NOT contagious.
Hemophilia i think... but i could be wrong
factor I (fibrinogen), factor II (prothrombin), factor III (tissue thromboplastin), factor IV (calcium), factor V (proaccelerin), factor VI (no longer considered active in hemostasis), factor VII (factor-vii), factor VIII (antihemophilicfactor), factor IX (plasma thromboplastincomponent; Christmas factor), factor X (stuart-factor-stuart-prower-factor), factor XI (plasma thromboplastinantecedent), factor XII (factor-xii), factor XIII (fibrin stabilizing factor).
Standards of care for Hemophilia vary greatly around the world and from one doctor to another there are variations of care. Most doctors agree treatment of Hemophilia is administration of anti-hemophiliac factor. The main treatment for hemophilia is called replacement therapy. Concentrates of clotting factor VIII (for hemophilia A) or clotting factor IX (for hemophilia B) are slowly dripped in or injected into a vein. These infusions help replace the clotting factor that's missing or low.
No,All are made in the liver except for Factor VIII (made by the vascular endothelium) and vonWillebrand's factor which is made in the endothelium (in the Weibel-Palade bodies), megakaryocytes (α-granules of platelets), and subendothelial connective tissue.