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Creatine kinase-MB (CK-MB), myoglobin, homocysteine, C-reactive protein (CRP), troponin T (cTnT), and troponin I (cTnI)

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What do you do when you suspected the people that dashed and dined on my sims agents?

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Can cigarette smoking lead to heart disease?

Cigarette smoking leads to heart disease. It contains numerous toxic agents along with nicotine. Nicotine stimulates the nicotinic receptors present in brain and in autonomous nervous system. So it excites the brain and both the sympathetic and parasympathetic nervous systems. It increases the blood pressure and make the wall of artery less elastic, so it hastens the process of atherosclerosis. Both of these are risk factors associated with myocardial infarction (heart attack).


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You use oedema reducing agents in stroke. Give general supportive care to the patient, including intravenous fluids. Then, two types of patho-physiology affect you with stroke. One is by way of infarction of the brain tissue and other is by way of bleeding inside the brain tissue. This can be confirmed by CAT scanning. You give blood thinning agents in case of infarction including low molecular weight heparin or heparin itself. Most of the patients eventually recover, partially or totally.


What has the author M I Gabriel Khan written?

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How are the mechanisms of action of class I and class III antiarrhythmic drugs in treating ventricular arrhythmia differents?

Class I antiarrhythmic drugs work by inhibiting the sodium channel. Class IA agents work by inhibiting open voltage-dependent Na+ channels. This will slow phase 0 and increase the length of the effective refractory period. Its effect is dependent on frequency. The drugs dissociate slowly from closed channels so when the frequency of the action potential is high, the drug can prevent the the Na+ channel from contributing to the action potential as it is still there. One example of a class IA agent is the procainamide which is used to treat ventricular arrhythmias when administered intravenously. Class IB agents work by blocking closed voltage-dependent Na+ channels. It is often used to treat ventricular arrhythmia after an acute myocardial infarction. An acute myocardial infarction often leads to anoxia so many Na+ channels will be inactiated and closed. These Na+ channels are hence susceptible to class IB agents. An example of a class IB agent is the lidocaine. Class III antiarrhythmic drugs work by blockign the outward K+ currents. This leads to the action potential belong prolonged. The QT interval will increase the the effective refractory period will also increase. One example of class III antiarrhythmic drug is the amiodarone. It inhibits the K+ channel, the inactivated Na+ channel, and the beta adrenoceptors.


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What are the drugs used in myocardial infarction?

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What has the author Lesia Anne Eva Stebelsky written?

Lesia Anne Eva Stebelsky has written: 'Patterns of prescribing practices in hypercholesterolemia, heart failure and post-myorcardial infarction: examining the use of angiotensin converting enzyme inhibitors and cholesterol lowering agents'