well it dies and then it smells like something that's dead
They both release carbon dioxide,but at different temperatures
The binding of neurotransmitters to receptors on the muscle membrane triggers the opening of ion channels, allowing sodium ions to enter the muscle cell. This influx of sodium ions initiates an action potential, leading to muscle contraction.
The solubility of sodium carbonate in water increases as the temperature rises. This means that more sodium carbonate can dissolve in water at higher temperatures compared to lower temperatures.
When sodium is pumped out of the cell by the sodium-potassium pump, it helps maintain the cell's resting membrane potential by generating an electrochemical gradient. This process also helps regulate cell volume and is essential for proper nerve and muscle function.
Sodium deficiency, known as hyponatremia, occurs when there is an imbalance of sodium levels in the blood. This can lead to symptoms such as lethargy, confusion, seizures, muscle cramps, and in severe cases, can be life-threatening. It is important to address the underlying cause of sodium deficiency and seek medical treatment if symptoms appear.
The melting point of sodium chloride is 801 0C.
When acetylcholine binds to the chemically gated ion channels on the plasma membrane of the muscle fiber, it causes these channels to open, allowing sodium ions to flow into the cell. This influx of sodium ions depolarizes the muscle fiber membrane, generating an action potential. The action potential then triggers the release of calcium ions from the sarcoplasmic reticulum, ultimately leading to muscle contraction.
Nothing
you get sodium chloride NaCl
It produces Sodium hydroxide and Hydrogen
Hyponatremia refers to a low blood concentration of sodium. Sodium is an essential electrolyte that helps maintain balance in bodily fluids and is important for nerve and muscle function. Symptoms of hyponatremia can include confusion, nausea, and muscle cramps.
When acetylcholine does not bind to muscle cells, it prevents the activation of nicotinic acetylcholine receptors at the neuromuscular junction. This failure to activate the receptors inhibits the influx of sodium ions, which is necessary for generating an action potential in the muscle fiber. Consequently, muscle contraction cannot occur, leading to muscle weakness or paralysis. This can happen in various conditions, such as myasthenia gravis or in the presence of certain toxins.