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Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
The sodium/potassium pump, the sodium leak channel and the potassium leak channel.
opening of sodium channels
Lidocaine is what is called an open sodium channel blocker. This means that it binds preferentially to sodium channels when they are in the open (letting sodium into the cell) state.
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Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
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through what can be seen as the domino effect. when depolarization opens sodium ion channel and that results to the opening of more sodium ion channels. this creates a positive feedback loop.
The acetylcholine diffuses across the synapse and binds to and activates nicotinic acetylcholine receptors on the motor end plate of the muscle cell. Activation of the nicotinic receptor opens its intrinsic sodium/potassium channel, causing sodium to rush in and potassium to trickle out.
a sodium channel blocker would (increase;decrease) serum sodium levels outside of normal resting nerve cells
The sodium/potassium pump, the sodium leak channel and the potassium leak channel.
The cell becomes depolarized
depolarization.
opening of sodium channels
Sodium enters the cell and causes depolarization. A small amount of potassium also leaves the motor end plate. This means binding of the neurotransmitter causes chemically gated sodium channels to open in the motor end plate.calcium
At rest sodium in the outside and potassium on the inside as action potential propagate along the axon, depolirization happens and sodium channel opens and allow sodium ions to flood into the neurone. A wave of deporization spread along the neuron, the neuron membrane contain specialised protein called channels. the channel from pore.