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An ion channel.
An action potential starts when sodium channels in a neuron end open and sodium ions rush is, depolarizing the neuron's membrane.
voltage gated ion channel
A temporary accumulation of sodium ions at the axon hillock which yields a voltage sufficient to open voltage-gated ion pores on the axon is what triggers an action potential.
Action potentials are how nerve impulses are transmitted from neuron to neuron. An action potential is formed when a stimulus to the nerve cell causes the membrane to depolarize and open all of its sodium ion channels toward the threshold potential.
An ion channel.
sodium potassium and calcium
factors that influences conductivities of action potential
An action potential starts when sodium channels in a neuron end open and sodium ions rush is, depolarizing the neuron's membrane.
Depolarization is the first event in action potential. During depolarization, the sodium gates open and the membrane depolarizes.
voltage gated ion channel
...repolarization
Voltage-gated channels are proteins in the cell membrane which open when stimulated by a voltage (an electrical signal). The voltage causes the channel to open, thereby allowing the entry or exit of whatever substance the channel relates to. An example of this the the voltage-gated sodium channels on neurons. When an action potential (a voltage), passes over the cell, it open these channels and allows sodium to enter the cell.
A temporary accumulation of sodium ions at the axon hillock which yields a voltage sufficient to open voltage-gated ion pores on the axon is what triggers an action potential.
Sodium ions are responsible for the rising phase of the action potential. This occurs when sodium channels open and sodium ions flow into the cell, causing depolarization.
Action potentials are how nerve impulses are transmitted from neuron to neuron. An action potential is formed when a stimulus to the nerve cell causes the membrane to depolarize and open all of its sodium ion channels toward the threshold potential.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.