A temporary accumulation of sodium ions at the axon hillock which yields a voltage sufficient to open voltage-gated ion pores on the axon is what triggers an action potential.
When an action potential reaches the end of a neuron, it triggers the release of neuotransmitters such as epinephrine (sympathetic) or achetylcholine (parasympathetic).
An ion channel.
depolarization
depolarization
No, calcium itself is not a neurotransmitter BUT it is highly important in the process of the action potential. The action potential triggers the influx of calcium at the end of the terminal bouton, causing the influx of Ca2+ into the cell and this triggers for the release of the neurotransmitter. :)
depolarization
When an action potential reaches the end of a neuron, it triggers the release of neuotransmitters such as epinephrine (sympathetic) or achetylcholine (parasympathetic).
despolarization
An ion channel.
depolarization
depolarization
No, calcium itself is not a neurotransmitter BUT it is highly important in the process of the action potential. The action potential triggers the influx of calcium at the end of the terminal bouton, causing the influx of Ca2+ into the cell and this triggers for the release of the neurotransmitter. :)
action potential propagating down the T tubule
The electrical current triggers an action potential at the neuromuscular junction, causing the muscle cells to depolarize and contract.
It creates an action potential
it triggers horrnyness
This is called action potential. Action potential is the change in electrical potential that occurs between the inside and outside of a nerve or muscle fiber when it is stimulated, serving to transmit nerve signals.