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The action potential will not generate if the sodium channels are kept closed.This is because the sodium channels are responsible for the dramatic rising phase of membrane depolarization that occurs when the threshold of activation is reached. As a membrane potential gradually depolarizes (which can occur for a variety of reasons such as neurotransmitter stimulation, mechanical deformation of the membrane, etc), that membrane potential gradually comes closer to that threshold of activation. Once that threshold is reached, the voltage gated sodium channels open and allow for a dramatic influx of sodium ions into the cell. This results in a rapid depolarization which is seen as the rising phase of that upward spike noted in an action potential. Without the ability to open these sodium channels we may reach the threshold of activation, but the actual action potential will not occur.
Sodium channels. A neuron's membrane potential may depolarize for many reasons (neurotransmitters, mechanical deflection, electrical synapse, etc). When that membrane depolarizes to the point of its threshold of activation, then voltage gated channels open up an allow an influx of sodium into the cell. This rapidly depolarizes the cell's membrane, causing that upward peak or rising phase to occur.
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
Yes
yes it's true.
Closed means just that...not open and unable to allow sodium ions to flow. Inactivated means that they do not respond to the stimulus. They could be open or closed, but do not receive the signal from the messenger. Also refractory.
The action potential will not generate if the sodium channels are kept closed.This is because the sodium channels are responsible for the dramatic rising phase of membrane depolarization that occurs when the threshold of activation is reached. As a membrane potential gradually depolarizes (which can occur for a variety of reasons such as neurotransmitter stimulation, mechanical deformation of the membrane, etc), that membrane potential gradually comes closer to that threshold of activation. Once that threshold is reached, the voltage gated sodium channels open and allow for a dramatic influx of sodium ions into the cell. This results in a rapid depolarization which is seen as the rising phase of that upward spike noted in an action potential. Without the ability to open these sodium channels we may reach the threshold of activation, but the actual action potential will not occur.
Rods and cones in the retina have photosensitive channels that open when a certain wavelength of light comes in contact. The open channels then either allow neurotransmitter or sodium to enter causing a chain reaction of the voltage gated Sodium Potassium pumps sending a signal down the bipolar neuron leading to the optic nerve.
Sodium channels. A neuron's membrane potential may depolarize for many reasons (neurotransmitters, mechanical deflection, electrical synapse, etc). When that membrane depolarizes to the point of its threshold of activation, then voltage gated channels open up an allow an influx of sodium into the cell. This rapidly depolarizes the cell's membrane, causing that upward peak or rising phase to occur.
nacl diffuse in plasma membrane because there are protein channels that allows certain ions to diffuse around the membrane, like sodium and chloride ions, please note that these channel proteins are selectively permeable meaning sodium channels only allow sodium to enter the cell and so on... urea diffuses into the pm the same way through facilitated diffusion of certain protein channels glucose diffuse into the pm as a part of secondary active transport, which means it uses ATP indirectly. it diffuses in the process called symport
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
At rest sodium in the outside and potassium on the inside as action potential propagate along the axon, depolirization happens and sodium channel opens and allow sodium ions to flood into the neurone. A wave of deporization spread along the neuron, the neuron membrane contain specialised protein called channels. the channel from pore.
Membrane receptors at a synapse are ligand-gated ion channels that open and allow sodium ions to flow into the neuron upon binding of the neurotransmitter ligand to generate an action potential in the neuron.
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
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Ions and large molecules
Yes