Brain liver heart
The tissues most severely affected by acute cyanide poisoning are the brain and the heart. This is because both organs have high oxygen demands and rely heavily on aerobic metabolism. Cyanide inhibits the enzyme cytochrome c oxidase in the electron transport chain, disrupting cellular respiration and leading to rapid cell death in these tissues.
Cyanide
cyanide
cyanide
cyanide
cyanide
Hydrogen peroxide (H2O2) is a compound that can attack oxygen-dependent tissues, causing oxidative damage. This can lead to cell injury and inflammation in tissues that rely on oxygen for their function.
No, artificial respiration would not save a person who has cyanide poisoning. Cyanide inhibits cellular respiration by blocking the enzyme cytochrome c oxidase in the mitochondria, preventing the cells from using oxygen. Therefore, providing artificial respiration would not be effective in delivering oxygen to the cells and reversing the effects of cyanide poisoning. Immediate medical intervention with antidotes such as hydroxocobalamin or sodium thiosulfate is crucial in treating cyanide poisoning.
Cyanide measures of whole blood, gastric contents, tissues, urine
Cyanide primarily affects tissues with high oxygen demands such as the brain and heart. It inhibits cellular respiration by blocking cytochrome c oxidase in the electron transport chain, leading to cellular hypoxia and eventually cell death.
Compounds that fit this description include hydrogen peroxide and peracetic acid. These chemicals are strong oxidizing agents that can quickly react with and damage oxygen-dependent tissues due to their oxidative properties.
Compounds like hydrogen peroxide and ozone are known to be fast-acting and attack oxygen-dependent tissues, such as bacterial cells or human cells. These compounds generate reactive oxygen species which can damage cellular components like DNA, proteins, and lipids, leading to cell death.