Brain, liver, heart
No, cyanide poisoning stop the respiration .
Maybe I think so a little probably not sometimes.
cyanide
Cyanide poisoning occurs because cyanide inhibits the enzyme cytochrome c oxidase, which is one of the enzymes that enable cells to use oxygen. Antidotes for cyanide poisoning tend to focus on binding the cyanide ion so that this inhibition stops. The liver is capable of metabolizing cyanide as well, to take care of any small residual amounts remaining.There are several ways to accomplish this. Nitrites and/or 4-dimethylaminophenol both convert hemoglobin to methemoglobin, which binds tightly with cyanide. This causes problems of its own, though ... methemoglobin cannot carry oxygen, and it's necessary to convert a lot of hemoglobin to methemoglobin to be sure there's an excess to bind all the cyanide, which leaves the person treated with methemoglobinemia ... which then must be treated separately (usually by treatment with methylene blue). Thiosulfates react with cyanide to convert it to thiocyanide, which is much less toxic. However, the reaction does not occur quickly in the body, so this treatment must be supplemented with one of the others (usually nitrates/4-DMAP). Cyanide is present in one form of vitamin B12, so treatment with the cyanideless form hydroxocobalamin will bind cyanide in the harmless cyanocobalamin. Cobalt ions in general will bind cyanide, but cyanocobalt complexes are themselves generally toxic, so this is not necessarily much of an improvement. It is fast, though, and is sometimes used (in the form of the dicobalt EDTA complex) in cases of severe cyanide poisoning. Administration with glucose helps mitigate the toxic effects of the cyanocobalt complexes.
It is important to avoid losses in the environment, contamination, poisoning of workers, thefts.
Brain liver heart
Liver, Spleen & Brain (Not the kidneys)
No, cyanide poisoning stop the respiration .
Mitochondria are the cellular organelles that would be affected by cyanide poisoning. Cyanide inhibits the enzyme cytochrome c oxidase, which is involved in the electron transport chain in the mitochondria. As a result, cells are unable to produce ATP, leading to cellular dysfunction and eventual cell death.
Vapors sniffed to counter the effects of cyanide poisoning.
Symptoms of cyanide poisoning in dogs may include difficulty breathing, seizures, vomiting, diarrhea, and sudden collapse. Immediate veterinary attention is crucial if poisoning is suspected.
Cyanide Poisoning.
Cyanide poisoning and a bullet.
Signs of cyanide poisoning in dogs may include difficulty breathing, seizures, vomiting, diarrhea, and sudden collapse. If you suspect your dog has been exposed to cyanide, seek immediate veterinary care.
The cyanide antidote is nitrite, specifically amyl nitrite or sodium nitrite, which helps to convert hemoglobin to methemoglobin, allowing it to bind cyanide. 2-PAM chloride (pralidoxime) is an antidote for organophosphate poisoning. Atropine is used to treat certain types of poisoning or overdose, but not specifically for cyanide poisoning.
Cyanide poisoning.
No, artificial respiration would not save a person who has cyanide poisoning. Cyanide inhibits cellular respiration by blocking the enzyme cytochrome c oxidase in the mitochondria, preventing the cells from using oxygen. Therefore, providing artificial respiration would not be effective in delivering oxygen to the cells and reversing the effects of cyanide poisoning. Immediate medical intervention with antidotes such as hydroxocobalamin or sodium thiosulfate is crucial in treating cyanide poisoning.