(I'm not sure if this answer is correct)
Here's the correct answer in better detail:
a. Generation of an action potential involves a transient increase in Na+ permeability, followed by restoration of Na+ impermeability, and then a short-lived increase in K+ permeability.
b. Propagation, or transmission, of an action potential occurs as the local currents of an area undergoing depolarization cause depolarization of the forward adjacent area.
It is called saltatory conduction. This describes the "jumping" of an action potential from node to node on a myelinated axon.
Action potentials are generated at the nodes of Ranvier during saltatory conduction. These nodes are the non-myelinated gaps found along the axon where the action potential can occur, allowing for faster transmission of the electrical signal down the nerve fiber.
The action potential travels in one direction because of the refractory period, which prevents the neuron from firing again immediately after an action potential has been generated. This ensures that the signal moves in a linear fashion along the neuron.
An action potential propagates unidirectionally along an axon because of the refractory period, which prevents the neuron from firing in the opposite direction immediately after an action potential is generated. This ensures that the signal travels in one direction, from the cell body to the axon terminal.
Unmyelinated tissue is substantially slower in conducting impulses along the axon. With myelinated axons, the action potential (impulse) jumps from node to node greatly increasing the speed of the impulse.
It is called saltatory conduction. This describes the "jumping" of an action potential from node to node on a myelinated axon.
a small myelinated axon
Action potentials are generated at the nodes of Ranvier during saltatory conduction. These nodes are the non-myelinated gaps found along the axon where the action potential can occur, allowing for faster transmission of the electrical signal down the nerve fiber.
myelinated, large diameter fibres
TRUE. Neurons with myelin (or myelinated neurons) conduct impulses much faster than those without myelin.
The action potential is generated when a stimulus causes a change in the electrical potential across the cell membrane, resulting in the opening of voltage-gated ion channels. This allows an influx of sodium ions, causing depolarization of the membrane and initiation of the action potential.
An action potential is not passively propagated down the axon. There have to be ion channels along the axon or else the action potential will gradually decay. So the the rate of that the action potential 'travels' is dependent on the passive property called the length constant of the axon (factor in capacitance, axon diameter) plus the density of ion channels.
Myelinated axons with a larger diameter will conduct action potentials the fastest due to saltatory conduction, where the action potential jumps from one node of Ranvier to the next, skipping the myelin-covered regions. Smaller-diameter and unmyelinated axons will conduct action potentials more slowly.
An action potential is generated at the axon hillock of a neuron, which is the region where the cell body (soma) transitions into the axon. This is where the concentration of voltage-gated sodium channels is highest, allowing for the initiation of the action potential.
Potassium.
it contracts
Myocardial contraction