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Cross-bridge formation that results in muscle contraction requires intracellular?
the major intracellular ion in crossbridge formation is what
What is the process of excitation contraction coupling?
cross bridge formation "Excitation-contraction coupling" connects muscle fiber excitation to the muscle fiber contraction (cross bridge formation). During contraction, myosin heads form cross bridges many times-with each cross bridge generating a small amount of tension in the muscle fiber.
ADP molecules on the surface of actin serve as active sites for the formation of cross bridges with molecules of?
ATP not ADP binds to actin-myosin and is cleaved by to ADP.
Atp allows what filaments to release and reset?
ATP allows actin and myosin filaments to release from each other during muscle contraction by assisting in the detachment of the myosin heads from actin. It also helps in resetting the myosin heads for the next contraction cycle by providing energy for the process of cross-bridge formation.
When the sarcomere is at rest what is covering the active sites on actin?
When the sarcomere is at rest, the active sites on actin are covered by tropomyosin molecules. Tropomyosin blocks the myosin-binding sites on actin, preventing cross-bridge formation and muscle contraction.
What best describes the events of excitation in excitation-contraction coupling?
cross bridge formation "Excitation-contraction coupling" connects muscle fiber excitation to the muscle fiber contraction (cross bridge formation). During contraction, myosin heads form cross bridges many times-with each cross bridge generating a small amount of tension in the muscle fiber.
How would the loss of acetylcholinesterase from the motor end affect skeletal muscle?
Acetylcholine release is necessary for skeletal muscle contraction, because it serves as the first step in the process, enabling the subsequent cross-bridge formation. A muscle's ability to contract depends on the formation of cross-bridges between myosin & actin filaments. A drug that blocks acetylcholine release would interfere with this cross-bridge formation and prevent muscle contraction
What is molecular muscular contraction?
1. ATP hydrolysis 2. Cross-bridge formation 3. Release of ADP and phosphate (p) 4. ATP binding; Cross-bridge dissociation
What describes the events of contraction in excitation-contraction coupling?
cross bridge formation "Excitation-contraction coupling" connects muscle fiber excitation to the muscle fiber contraction (cross bridge formation). During contraction, myosin heads form cross bridges many times-with each cross bridge generating a small amount of tension in the muscle fiber.
Cross-bridge formation that results in muscle contraction requires intracellular?
the major intracellular ion in crossbridge formation is what
What is the process of excitation contraction coupling?
cross bridge formation "Excitation-contraction coupling" connects muscle fiber excitation to the muscle fiber contraction (cross bridge formation). During contraction, myosin heads form cross bridges many times-with each cross bridge generating a small amount of tension in the muscle fiber.
How does blocking the activity of acetylcholinesterase affect skeletal muscle?
Acetylcholine release is necessary for skeletal muscle contraction, because it serves as the first step in the process, enabling the subsequent cross-bridge formation. A muscle's ability to contract depends on the formation of cross-bridges between myosin & actin filaments. A drug that blocks acetylcholine release would interfere with this cross-bridge formation and prevent muscle contraction
ADP molecules on the surface of actin serve as active sites for the formation of cross bridges with molecules of?
ATP not ADP binds to actin-myosin and is cleaved by to ADP.
Atp allows what filaments to release and reset?
ATP allows actin and myosin filaments to release from each other during muscle contraction by assisting in the detachment of the myosin heads from actin. It also helps in resetting the myosin heads for the next contraction cycle by providing energy for the process of cross-bridge formation.
Why are some bridges supported by rolles at one side?
Thermal expansion and contraction can put huge stresses into a large structure such as a bridge. To minimise this effect, the rollers are designed to allow the end of the bridge to move with the expansion and contraction.
What effect do calcium slow channels haveon shortening or lengthing the contraction time of the heart?
Calcium slow channels play a crucial role in cardiac muscle contraction by allowing calcium ions to enter the cell. Activation of these channels leads to an increase in intracellular calcium levels, which ultimately shortens the contraction time of the heart by promoting cross-bridge formation between actin and myosin. In contrast, inhibition of calcium slow channels would lead to a lengthening of contraction time due to reduced calcium availability for muscle contraction.
When the sarcomere is at rest what is covering the active sites on actin?
When the sarcomere is at rest, the active sites on actin are covered by tropomyosin molecules. Tropomyosin blocks the myosin-binding sites on actin, preventing cross-bridge formation and muscle contraction.
