Motor end plate of a cell is the dendritic portion that have cholinergic receptors to receive neurotransmitters in order to propagate an action potential.
The point at which a motor neuron synapses with a muscle fiber's motor end plate is called the neuromuscular junction. This is where the nerve impulse is transmitted from the motor neuron to the muscle fiber, leading to muscle contraction.
When the neurotransmitter acetylcholine binds to the motor end plate, it triggers the opening of sodium channels in the muscle cell membrane. This influx of sodium ions leads to depolarization of the cell, creating an action potential that propagates along the muscle fiber, ultimately leading to muscle contraction.
When a neurotransmitter binds to its receptor on the motor endplate, it triggers the opening of ion channels in the postsynaptic membrane. This allows for the influx of ions, typically leading to depolarization of the muscle cell membrane and initiation of a muscle action potential. Subsequently, this leads to contraction of the muscle fiber.
Acetylcholine receptors are located on the motor end plate of the muscle cell membrane. This specialized region is where the nerve cell communicates with the muscle cell, allowing for the initiation of muscle contraction in response to acetylcholine binding to its receptors.
Acetylcholine receptor is present on the sarcolemma of the muscle cells. This receptor is responsible for transmitting the signal to initiate muscle contraction when acetylcholine binds to it at the neuromuscular junction.
The point at which a motor neuron synapses with a muscle fiber's motor end plate is called the neuromuscular junction. This is where the nerve impulse is transmitted from the motor neuron to the muscle fiber, leading to muscle contraction.
Curare poisoning affects the motor end plate. Curare blocks nicotinic acetylcholine receptors on muscle cells at the neuromuscular junction, preventing the binding of acetylcholine and leading to muscle paralysis.
motor end plate
The receptors found on the end of the motor end plate are called nicotinic acetylcholine receptors. These receptors respond to the neurotransmitter acetylcholine released from the motor neuron and play a crucial role in initiating muscle contraction.
Acetylcholine is the primary neurotransmitter released at the motor end plate, where it binds to receptors on muscle cells to initiate muscle contraction.
The neuromuscular junction consists of the motor neuron terminal, synaptic cleft, and motor end plate on the muscle fiber. When an action potential reaches the motor neuron terminal, it triggers the release of acetylcholine into the synaptic cleft. Acetylcholine then binds to receptors on the motor end plate, leading to muscle contraction.
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The folded region of the sarcolemma at the neuromuscular junction is called the motor end plate. It is responsible for receiving signals from the motor neuron and initiating muscle contraction. The motor end plate is rich in acetylcholine receptors, which play a key role in neurotransmission at the neuromuscular junction.
A deficiency of ACh receptors in a motor end plate would cause muscle weakness or paralysis. This condition is called myasthenia gravis.
Neuromuscular Junction
When acetylcholine binds to receptors at the motor plate, this binding opens ligand-gated ion channels on the motor end plate, ions diffuse through the open ligand gated ion channels, and the flow of ions causes the motor end plate to reach threshold and an action potential is generated at the motor end plate.
motor end plate