The energy of the high energy molecules used for every time 2 high energy electrons move down the chain causes the H+ ions to move to the matrix.
The opening of sodium voltage-gated channels in the neuronal membrane is caused by changes in the electrical charge across the membrane, known as membrane potential. When the membrane potential reaches a certain threshold, the channels open, allowing sodium ions to flow into the neuron and generate an action potential.
Opening sodium channels in the axon membrane allows sodium ions to flow into the cell, depolarizing the membrane and generating an action potential. This action potential then travels down the axon to facilitate neuronal communication and signal transmission.
The electron transport chain converts energy stored in hydrogen ions and various other substances formed in early cellular respiration to produce high energy ATP in mitochondria. Mitochondria contain both an inner and an outer membrane, and it is along the inner membrane that the actual reactions of the chain occur. Inside the inner membrane a surplus of hydrogen ions is created that produces a concentration gradient across the membrane to the intermembrane space. This gradient causes a force that pushes hydrogen ions out of the innermost matrix and into the intermembrane space. This exchange occurs through special proteins called ATP synthase that convert low energy ADP into high energy ATP whenever a hydrogen ion is sent through one. When all is said and done, the excess electrons and hydrogen are bonded to oxygen to form water molecules.
When a neurotransmitter binds to its receptor on the motor endplate, it triggers the opening of ion channels in the postsynaptic membrane. This allows for the influx of ions, typically leading to depolarization of the muscle cell membrane and initiation of a muscle action potential. Subsequently, this leads to contraction of the muscle fiber.
Transmembrane channels in target cells are typically formed by specialized proteins that span the cell membrane. These proteins create a passageway for specific ions or molecules to move across the membrane, allowing for communication and transport between the cell's interior and exterior environments. The opening and closing of these channels can be regulated by various factors, including voltage changes, ligand binding, or mechanical force.
The energy of the high energy molecules used for every time 2 high energy electrons move down the chain causes the H+ ions to move to the matrix.
The energy of the high energy molecules used for every time 2 high energy electrons move down the chain causes the H+ ions to move to the matrix.
The opening of sodium voltage-gated channels in the neuronal membrane is caused by changes in the electrical charge across the membrane, known as membrane potential. When the membrane potential reaches a certain threshold, the channels open, allowing sodium ions to flow into the neuron and generate an action potential.
An area of the inner mitochondrial membrane becomes positively charged as a result of the electron transport chain process during cellular respiration. During this process, protons are pumped across the inner membrane, creating an electrochemical gradient with a higher concentration of protons in the intermembrane space compared to the mitochondrial matrix. This results in a positively charged intermembrane space and a negatively charged matrix.
Binding of acetylcholine to nicotinic acetylcholine receptors opens ion channels that allow both sodium and potassium ions to permeate the membrane. This causes depolarization of the membrane potential, leading to an excitatory response in the cell.
Opening or closing of ion channels at one point in the membrane produces a local change in the membrane potential, which causes electric current to flow rapidly to other points in the membrane.
Cell membrane depolarization is caused by the influx of positively charged ions, such as sodium ions, through ion channels in the membrane. This influx of positive charge reduces the voltage difference across the membrane, leading to depolarization.
depolarization of the presynaptic membrane due to an arriving action potential
NA plus channels open in response to a change in the membrane potential, causing the channel to undergo conformational changes that lead to its opening. This change in membrane potential can be initiated by various stimuli, such as neurotransmitter binding or depolarization of the cell.
Opening sodium channels in the axon membrane allows sodium ions to flow into the cell, depolarizing the membrane and generating an action potential. This action potential then travels down the axon to facilitate neuronal communication and signal transmission.
The electron transport chain converts energy stored in hydrogen ions and various other substances formed in early cellular respiration to produce high energy ATP in mitochondria. Mitochondria contain both an inner and an outer membrane, and it is along the inner membrane that the actual reactions of the chain occur. Inside the inner membrane a surplus of hydrogen ions is created that produces a concentration gradient across the membrane to the intermembrane space. This gradient causes a force that pushes hydrogen ions out of the innermost matrix and into the intermembrane space. This exchange occurs through special proteins called ATP synthase that convert low energy ADP into high energy ATP whenever a hydrogen ion is sent through one. When all is said and done, the excess electrons and hydrogen are bonded to oxygen to form water molecules.
The first step for nerve impulse generation is the depolarization of the cell membrane, which is triggered by a stimulus. This depolarization causes a change in the electrical charge of the cell membrane, leading to the opening of ion channels and the initiation of an action potential.