I think that with the destruction of myelin (produced by Oligodendrites in the CNS or the Schwann cells in the PNS that are glial cells that speed up the signal), the signal will be slower or even lost.
A disease that effects the myelin sheath is multiple sclerosis (MS). The function of the myelin sheath is to act as an insulator for the nerve cell. It's kind of like the rubber casing found on the outside of wires to protect the electron movement inside and allow us not to get shocked. MS causes the destruction of the myelin sheath which causes it to scar and harden and as a result we short circuit in our body. This is equivalent to peeling off some of the rubber casing off a wire and then plugging the wire into a socket. Some results of MS are: -double vision -speech impairment -jerky limbs -paralyzed muscles.
Gaps in the myelin sheath can result in reduced conduction velocity of nerve impulses, leading to impaired communication between neurons. This can manifest as symptoms such as weakness, numbness, and coordination issues. Conditions like multiple sclerosis can cause damage to the myelin sheath, resulting in these gaps.
Myelin insulation insulates nerve fibers by wrapping around them and forming a protective sheath. This myelin sheath helps to increase the speed of nerve impulse conduction along the nerve fibers.
Diseases that destroy the myelin sheath, such as multiple sclerosis, disrupt the ability of nerve cells to efficiently transmit signals. This results in impaired communication between the brain and muscles, leading to weakness and paralysis. The myelin sheath is crucial for protecting and insulating nerve fibers, so its destruction can significantly impact motor function.
The fatty tissue that covers axons and speeds up impulses is called myelin. Myelin acts as an insulating layer, which helps to increase the speed and efficiency of nerve impulse transmission along the axon.
The reason for the destruction of myelin in GBS is unknown, although it is thought that the underlying problem is autoimmune in nature
MS
Muscular sclerosis is hardening and degeneration of the myelin sheath. Muscular sclerosis Multiple sclerosis is a disorder marked by destruction of the myelin sheath on neurons in the CNS and replacement with hard scar tissue.
Demyelinating diseases such as Multiple Sclerosis, Krabbe's Disease, and Guillain-Barre Syndrome involve degeneration of the myelin sheath. In these conditions, movement is limited to minimal as nerve conduction is reduced due to the loss of efficacy of the myelin.
A disease that effects the myelin sheath is multiple sclerosis (MS). The function of the myelin sheath is to act as an insulator for the nerve cell. It's kind of like the rubber casing found on the outside of wires to protect the electron movement inside and allow us not to get shocked. MS causes the destruction of the myelin sheath which causes it to scar and harden and as a result we short circuit in our body. This is equivalent to peeling off some of the rubber casing off a wire and then plugging the wire into a socket. Some results of MS are: -double vision -speech impairment -jerky limbs -paralyzed muscles.
The myelin sheeth helps keep signals (like electrical impulses) within the axon so that they can get to the cell body (of the neuron). This is necessary for muscles to contract (to move). If myelin is lost the signal or "electrical impulse" is less and less which has an affect on muscle function because there isn't enough signal to stimulate or "tell" the muslce to move.
The autoimmune disease that results in the destruction of myelin sheaths is called multiple sclerosis (MS). This condition leads to disruption in the transmission of signals along the nerves, causing a range of symptoms including weakness, numbness, vision problems, and difficulties with coordination and balance.
No, myelin sheaths actually speed up nerve impulses by increasing the speed of signal transmission along the nerve fibers. This is due to the insulation effect of myelin, which helps to prevent signal loss or leakage.
Gaps in the myelin sheath can result in reduced conduction velocity of nerve impulses, leading to impaired communication between neurons. This can manifest as symptoms such as weakness, numbness, and coordination issues. Conditions like multiple sclerosis can cause damage to the myelin sheath, resulting in these gaps.
They will slow down
Myelin is composed of about 80% lipid and about 20% protein. Some of the proteins that make up myelin are myelin basic protein (MBP),myelin oligodendrocyte glycoprotein (MOG), and proteolipid protein (PLP). Myelin is made up primarily of a glycolipid calledgalactocerebroside. The intertwining of the hydrocarbon chains of sphingomyelin serve to strengthen the myelin sheath.There are quite a few disease (multiple sclerosis, optic neuritis, transverse myelitis..) involved in the destruction of the axon myelin sheath also known as myelin sheath.http://en.wikipedia.org/wiki/Myelin
The interaction of the destruction of the myelin sheaths and remyelination is not yet fully understood. The lesions and scarring of the nerve tissue seem to mainly occur in the CNS, with the autoimmune process largely ignoring the peripheral nervous system.