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Genes involved in the production of abnormal red blood cells may have mutations in their DNA sequence, leading to changes in the protein structure that result in abnormal red blood cells. These mutations can affect genes like HBB (hemoglobin beta), which is important for normal hemoglobin production, leading to conditions like sickle cell anemia.
yes - no its not because a sickle cell is basically a diseased cell that is shapped like a sickle.. a disease in bloodcell - it cannot get as much oxygen as healthy blood cells
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This is inherited on the X chromoseome and is recessive. This means a female (who has XX chromosome pair) can only express the disease if she carries the trait on both chromosomes. This is because of the recessive nature of the gene. A male can only inherit the disease from his mother. This is he must have obtained his Y chromosome from his father so he obtains the X chromosome from his mother. A male cannot carry the gene without expressing it as he doesnt have another X chromosome to supress the recessive gene. ^ This answer is biologically incorrect because Sickle cell Anemia is actually autosomal recessive, not sex-linked recessive; this means that males and females are affected equally and it is NOT carried on the X chromosome. In order for the trait to show up in an individual, BOTH parents must be carriers of the disease (at least 1 sickle cell gene must be present).
Not sure what HBB airsoft is, do you have the acronym correct. I know that GBB stands for Gas Blowback.
The HBB gene, which codes for the beta globin protein, is located on chromosome 11.
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Beta-thalassemia is typically caused by mutations in the HBB gene located on chromosome 11. Common mutations include point mutations, deletions, or insertions in the HBB gene, leading to reduced or absent production of beta-globin chains in hemoglobin. These mutations disrupt the normal structure and function of hemoglobin, resulting in anemia and other symptoms associated with beta-thalassemia.
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