Vitamin K corrects prothrombin in the setting of biliary obstruction but not of hepatocellular damage.
Vitamin K
K
prothrombin
Vitamin K is essential for the synthesis of prothrombin and thrombin. It acts as a cofactor in the carboxylation of specific glutamic acid residues on these proteins, which is crucial for their activity in the blood coagulation process. Without adequate vitamin K, the synthesis of functional prothrombin is impaired, leading to issues with blood clotting.
Jaundice
Vitamin K is responsible for the clotting of blood..
Potatoes are rich in vitamin D and they are safe to eat during jaundice. This includes sweet potatoes and yams.
Vitamin K deficiency is typically measured by assessing levels of prothrombin time (PT) and activated partial thromboplastin time (aPTT) in the blood. Additionally, measurement of vitamin K-dependent clotting factors, such as Factor II, VII, IX, and X, can also be used to diagnose deficiency. Specialized tests to directly measure vitamin K levels in the blood can also be performed.
A) Albumin B) GI mucosa C) Mucopolysaccharides D) Prothrombin
vitamin K, or, when rapid reversal is needed (such as in case of severe bleeding), with prothrombin complex concentrate.
Jaundice and carotenemia are the most common causes of yellow skin. Jaundice is accumulation of bilirubin which is a bile pigment and carotenemia is the accumulation of carotene; the precursor of vitamin A.
Jaundice from the accumulation of bile and nutritional deficiencies-bruising from lack of vitamin K, bone pain from lack of vitamin D, night blindness from lack of vitamin A, and skin rashes, lack of vitamin E.