The average elderly man or woman of around 65 yeas of age, has a negative calcium balance and a bone mass which is gradually depleting (osteoporosis). In older postmenopausal women, who have been in their menopause for at least five years or more, evidence suggests that the effectiveness of a higher intake of calcium to ward off or delay bone depletion, is at best unclear or has no effect. Exercise in youth is a factor which improves bone density in old age. Evidence also suggests that a calcium supplementation does not prevent bone loss for lactating women who breast-feed their babies and only slightly enhances the gain in bone density after the children are weaned. Supplementation with calcium citrate malate has been seen to be more effective than supplementation with calcium carbonate, depending on the doses tested. Calcium intake needs to be supplemented with vitamin D3 (cholecalciferol) in order to be most effective, rather than with just calcium.
the bone contain calcium salt whcih are less reactive than the calcium ions found in the blood.
Bone is made up mostly of calcium phosphate. There are many other different components to bone which are not fully understood, such as collagen and other organic chemicals. The molecular formula of calcium phosphate is Ca3(PO4)2. This is made of three Ca2+ ions and two PO43- ions.
Bone
hydroxyapatite
Parathyroid hormone provides a powerful mechanism for controlling extracellular calcium and phosphate concentrations by regulating intestinal reabsorption, renal secretion, and exchange between the extracellular fluid and bone of these ions. Also, Calcitonin, which is a peptide hormone secreted by the thyroid gland, tends to decrease plasma calcium concentration and usually has effects that are opposite to those of the parathyroid hormone.
In general, it increases calcium in the blood by increasing bone resorption. The exception is when pth is given in spiked pulses, which is the case in some medications (forteo, a pth analogue) which can paradoxically decrease bone resorption.
Osteoporosis is the name for low bone density.
The parathyroid glands (4 of them) secrete parathyroid hormone to increase blood calcium. When the parathyroid glands detect low blood calcium levels, they secrete more parathyroid hormone, which causes the bones to release calcium to the bloodstream, thereby increasing blood calcium levels.
The hormone Calcitonin has the effect of reducing blood calcium levels. The hormone comes from the thyroid gland and works in basically three ways. 1. It works to slow absorption of calcium by the intestine. 2. It also inhibits the osteoclast (cells in bone that break down old bone tissue) that raise blood calcium levels. 3. It causes osteoblast (cells in bone that lay down new bone matrix) to form new bone. This explanation is at it's simplest form of course.
A bone density test employs x-rays to measure the amount of calcium and other bone minerals are retained with the bone segment. A bone density test is used to diagnose osteoporosis.
Adequate calcium intake is necessary for strong bones and can help decrease the risk of fractures. People whodonot getenough calcium in their diets can take a calcium supplement. Exercise can help strengthen bones by increasing bone density
it takes calcium! it takes calcium!
A person should eat their normal diet before the bone density test. The only change that should be made to a diet is calcium supplements. Calcium supplements should not be taken before a bone density test.
Osteoclast
Osteoporosis is a bone disorder characterized by reduced bone mass and density. This is caused by inadequate amounts of calcium in the bones.
Yes your body does need calcium because if your body doesn't have calcium it would decrease your bone strength.
Osteoclasts