Defect in a simple cell: In a simple cell current cannot be produced for long time and Its is not easy to carry from one place to other because liquid electrolyte (dilute sulfuric acid) is used in it.
exacted from sir, Supran Acharya
Cancerous cells can have both genetic defects, such as mutations in key oncogenes or tumor suppressor genes, and functional defects, such as uncontrolled proliferation and evasion of cell death mechanisms. These defects allow cancer cells to grow uncontrollably and spread to other parts of the body.
Immunoglobulin deficiencies are the result of congenital defects affecting the development and function of B lymphocytes (B-cells). There are two main points in the development of B-cells when defects can occur. First.
Neonatal adrenoleukodystrophy , infantile Refsum disease , and hyperpipecolic acidemia involve defects in the cytoplasm organelles of cells called the peroxisomes
It can cause defects to the fetus
It breaks down the muscle cells, which leads to muscle weakness. Causing defects in the muscle proteins and the death of muscle cells and tissue.
The technique which involves withdrawing and analyzing cells in amniotic fluid and detects genetic and other birth defects is called amniocentesis. This option is offered to most pregnant women but it is there choice to have it done.
Defects.
Latent defects are not obvious and are not easily discoverable while patent defects are obvious.
It depends on the age of the person. There are more mutations and defects in eggs as the woman ages. However, as men are constantly producing more sperm cells they will inevitably produce more cells that have mutations. The percentage of sperms cells that have mutations will increase as the man ages.
Amniocentesis is the test carried out during 15 to 20 weeks into pregnancy to analyze fetal cells for genetic defects. It provides a nearly 100% accuracy in identifying a variety of genetic conditions and chromosomal abnormalities.
what the defects of timber
Intracorpuscular defects refer to abnormalities or dysfunctions within red blood cells themselves, often resulting in conditions like hemolytic anemia. These defects can arise from genetic mutations affecting hemoglobin synthesis, membrane structure, or metabolic pathways within the cells. Common examples include sickle cell disease and thalassemia, where the structural integrity or function of hemoglobin is compromised. These conditions lead to reduced red blood cell lifespan and various clinical symptoms associated with anemia.