It blocks the nicotinic cholinergic receptors on the muscle that normally bind the acetylcholine released by the motor neuron.
Curare is a plant-derived poison that blocks nicotinic acetylcholine receptors at the neuromuscular junction, leading to muscle relaxation and paralysis. This can result in impaired breathing and death if respiratory muscles are affected.
Curare is a competitive inhibitor of acetylcholine. It fights with acetycholine for postsynaptic receptors.
Muscle tissue is responsible for allowing the body to move by stretching and contracting. Muscles are made up of muscle fibers that can shorten (contract) and lengthen (stretch) to produce movement. Skeletal muscles, smooth muscles, and cardiac muscles are the three main types of muscle tissue in the body.
Curare acts by blocking nicotinic acetylcholine receptors at the neuromuscular junction, preventing the binding of acetylcholine to these receptors. This leads to muscle paralysis since acetylcholine is unable to initiate muscle contraction.
skeletal muscle tissue,cardiac muscle tissue smooth muscle tissue
It creates an action potential
Muscle tissue is deep to epidermal tissue. Epidermal tissue is superficial to muscle tissue.
It breaks down the muscle cells, which leads to muscle weakness. Causing defects in the muscle proteins and the death of muscle cells and tissue.
Curare poisoning is caused by the ingestion or injection of curare, a plant-derived toxin commonly used as a muscle relaxant. Symptoms of curare poisoning include paralysis, respiratory failure, and potentially death if left untreated. Treatment involves supporting vital functions and administering antidotes like cholinesterase inhibitors.
Curare reduces the response to nerve stimulation because it acts as a neuro-muscular blocking agent, or muscle relaxant. It is also known as tubocurarine.
Lactic acid and oxygen debt
Curare acts by blocking the transmission of nerve impulses at the neuromuscular junction, leading to muscle paralysis. It does this by binding to and blocking nicotinic acetylcholine receptors on the post-synaptic membrane, preventing the action potential from propagating across the muscle cell membrane.