It creates an action potential
Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
it is an alpha-toxin that binds to acetylcholine binding sites on the postsynaptic cell membrane, which prevents the acetylcholine from acting. Curare blocks synaptic transmission by preventing neural impulses to flow from neuron to neuron. It does allow the action potential to travel in the axon, it just doesn't pass it on to the dendrite.
Depends on disease
Archibald Ross McIntyre has written: 'The mechanism of the action of extract of the posterior lobe of the pituitary body on the secretion of urine in the unanaesthetized dog' -- subject(s): Physiological effect, Pituitary extract, Urination, Dogs, Physiology 'Curare' -- subject(s): Curare
Ether blocked the impulse transmission.
what effect does the drug quabain have on neuron
It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,and it can mask digitalis toxicity.
A synapse and an action potential have a flip-flopping cause and effect relationship, in that an action potential in a presynaptic neuron initiates a release of neurotransmitters across a synapse, which can then subsequently potentially trigger an action potential in the axon of the postsynaptic neuron, which would then cause release of neurotransmitters across a following synapse.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
They both decrease action potential duration, but TTX is the only one that decreases the maximum rate of depolarization.
It makes the muscle totally relax as it blocks the action potential in the nerves.
It blocks the sodium channels that are required to create action potential in the muscles to make them contract.