It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,
and it can mask digitalis toxicity.
Ether can enhance the excitability of nerve cell membranes, leading to a decrease in the threshold for action potential generation. This can result in an increase in the frequency and amplitude of action potentials.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
No, neurotransmitters that depress the resting potential are called inhibitory neurotransmitters. Excitatory neurotransmitters have the opposite effect, causing depolarization and increasing the likelihood of an action potential.
Hypokalemia (low potassium levels) can lead to a more negative resting membrane potential in cells. This enhances the threshold for depolarization and can result in muscle weakness, cramping, and cardiac arrhythmias due to impaired cell signaling.
Hypocalcemia
It creates an action potential
The MOST serious effect would be death.
A synapse and an action potential have a flip-flopping cause and effect relationship, in that an action potential in a presynaptic neuron initiates a release of neurotransmitters across a synapse, which can then subsequently potentially trigger an action potential in the axon of the postsynaptic neuron, which would then cause release of neurotransmitters across a following synapse.
Ether can enhance the excitability of nerve cell membranes, leading to a decrease in the threshold for action potential generation. This can result in an increase in the frequency and amplitude of action potentials.
They both decrease action potential duration, but TTX is the only one that decreases the maximum rate of depolarization.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
It makes the muscle totally relax as it blocks the action potential in the nerves.
It blocks the sodium channels that are required to create action potential in the muscles to make them contract.
A negative effect is any result of an action which is perceived to be detrimental. This means its a subjective opinion. What might be negative to you may be positive to someone else.
Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
No, neurotransmitters that depress the resting potential are called inhibitory neurotransmitters. Excitatory neurotransmitters have the opposite effect, causing depolarization and increasing the likelihood of an action potential.