is the tension or stress developed in the wall of the
left ventricleduring ejection. In other words, it is the end
Loadagainst which the heart contracts to eject blood.
Afterload
Afterload
The systemic arteries provide afterload for the left ventricle, while the pulmonary arteries provide afterload for the right ventricle. Afterload refers to the resistance that the ventricles must overcome to eject blood during systole.
afterload
Morphine decrease cathecolamines therefore decreases afterload.
It decreases preload and afterload as a result of the dilation in the venous and arterial vasculature from the nitric oxide.
Decreased afterload occurs when the resistance the heart must overcome to eject blood is reduced. This can be caused by factors such as vasodilation, which decreases systemic vascular resistance, or conditions like sepsis that lead to widespread blood vessel dilation. Additionally, medications such as ACE inhibitors or nitrates can also lower afterload by relaxing blood vessels. Ultimately, decreased afterload facilitates easier ventricular ejection, improving cardiac output.
Yes
Back pressure exterted by arterial blood
Yes, stroke volume is inversely proportional to afterload. An increase in afterload, such as from increased vascular resistance, can lead to a decrease in stroke volume due to the additional pressure the heart has to work against to eject blood. Conversely, decreasing afterload can help increase stroke volume.
ACE inhibitors primarily affect afterload by causing vasodilation, which reduces systemic vascular resistance. This action can lower blood pressure and decrease the workload on the heart. While they may have some indirect effects on preload by reducing fluid retention, their main impact is on afterload reduction.
it decreases blood volume and preload