0
Why did the rough strain and heat killed smooth strain kill the miceAsk us anything?
The rough strain of bacteria is non-virulent and does not cause disease, while the smooth strain is virulent due to its protective capsule. When the heat-killed smooth strain was introduced to the mice, it released its genetic material, which could be taken up by the rough strain through a process called transformation. This allowed the rough strain to acquire the virulence factors from the smooth strain, making it capable of causing disease and ultimately killing the mice. This phenomenon demonstrated the principle of bacterial transformation and the transfer of genetic traits.
What else can I help you with?
What is the strain?
The Andromeda strain is what is known as sweet sciency magic.
When was No Soul No Strain created?
No Soul No Strain was created in 415.
When was The Deviant Strain created?
The Deviant Strain was created in 2005.
What does Ferine strain mean?
Ferine means wild; untamed. Strain used with ferine means song. So ferine strain is 'wild song'.
What are the ratings and certificates for The Strain Andromeda - 1992?
The Strain Andromeda - 1992 is rated/received certificates of: Singapore:PG
When Fredrick griffith injected mice with a mixture of both heat killed smooth strain bacteria and live rough strain bacteria an unexpected result occured this result was?
The unexpected result was that the previously harmless rough strain bacteria were able to take up genetic material from the heat-killed smooth strain bacteria, becoming virulent. This demonstrated the concept of bacterial transformation and laid the foundation for understanding DNA as the genetic material.
What is transformed from rough strain bacteria to smooth strain cells?
The process of transforming rough strain bacteria into smooth strain cells involves transferring genetic material - specifically a capsule gene - from a smooth strain to the rough strain. This genetic transfer results in the expression of a protective capsule on the surface of the rough strain cells, converting them into smooth strain cells with enhanced virulence.
What are the release dates for The Strain - 2014 Gone Smooth 1-3?
The Strain - 2014 Gone Smooth 1-3 was released on: Canada: 2014 USA: 2014
What are 4 steps in Griffith's experiments?
S Strain ---> inject into mice ---> Mice Die R Strain ---> inject into mice ---> Mice Live Griffith was able to kill bacteria by heating them. He observed that heat-killed S Strain bacteria injected into mice did not kill them. When he S Strain (heat Killed) ---> Injected into mice ---> Mice Live S Strain ( heat Killed) + R Strain (Live) ---> Injected into mice ---> Mice Die
What happens when heat-killed S-strain pneumococcus were injected into a mouse along with live R-strain pneumococcus?
A very important characteristic about bacteria actually created live S-strain to exist in the dead injected mouse. Bacteria is competent, in biological terms, meaning that cells can take up "naked" DNA from it's environment. Knowing that fact, when the heat-killed S-strain pneumoccus mixed with the live R-strain, the R-strain took in the DNA, therefore inheriting the deadly coating of the S-strain. So the R became S, and killed the mouse.
Did you strain your ?
Did you strain your back when you were moving? Can you pinpoint where the pain is? Is there anything that makes it better or worse?
Avery macleod and mccarty found transformed live rough strain bacteria to smooth strain cellds and therefore was genetic material?
A. Dna
The strain of bacteria that caused pneumonia grew into what colonies on culture plates?
The disease-causing strain of bacteria grew into smooth colonies on culture plates, whereas the harmless strain produced with rough edges.
What important question remained unanswered by griffith experiment?
Assuming you mean the Frederick Griffith bacterial experiment, the question left unanswered was how the rough strain (less harmful) bacteria transformed into the smooth strain (lethal) bacteria. When he injected live rough strain bacteria, the mouse lived. When he injected live smooth strain bacteria, the mouse died. But if dead smooth strain bacteria was injected, the mouse lived. So if either live rough strain or dead smooth strain could be injected without killing the mouse, then it would stand to reason that one could inject both, the mouse should live. But the mouse died. So he figured the rough strain was somehow taking on the characteristics of the smooth strain bacteria, perhaps by being in close proximity to dead smooth strain bacteria, but he didn't know why. We now know that the smooth strain DNA was somehow getting grafted into the rough strain bacteria and making it able to create a coating which prevented the immune system (of the mouse in this case) from killing it. In case you mean the John Howard Griffin racial change experiment, the question left unanswered was how to stop racism. While his experiment was not completely successful, he gained a number of insights into the experiences of men of color in America. He was able to speak to people in both groups, but yet, he was not able at that point to get them to reconcile with each other.
What are two ways that rough bacteria differ from smooth bacteria?
Streptococcus pneumoniae bacteria is found in two types: a type III-S (smooth) and type II-R (rough) strain. The smooth strain covers itself with a polysaccharide capsule that protects it from the host's immune system, resulting in the death of the host, while the II-R strain doesn't have that protective capsule and is defeated by the host's immune system.The smooth type has a "coat" that prevents it from attacks from the host's immune system and it is very virulent.
What strain of bacteria that caused pneumonia grew into?
Some Biology books may say something different but mine is Prentice Hall Biology and its page 287. The answer to this would be smooth colonies. :) so if your ever looking for this answer hope it works for you!
How did Griffith's experiments show that a hereditary factor was involved in bacterial transformation?
Griffith's experiments demonstrated that a hereditary factor was involved in bacterial transformation through the use of two strains of Streptococcus pneumoniae: a virulent smooth strain and a non-virulent rough strain. When he injected mice with heat-killed smooth bacteria mixed with live rough bacteria, the mice developed pneumonia and died, indicating that the rough bacteria had somehow transformed into the virulent smooth form. This transformation suggested the presence of a "transforming principle," which later researchers identified as DNA, thus showing that genetic information could be transferred between bacteria.
