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When acetylcholine diffuses across the synaptic cleft and binds to receptors on the folded sarcolemma it initaly causes what channels to open in the sarcolemma?
Acetylcholine binding causes nicotinic acetylcholine receptors on the folded sarcolemma to open, allowing the influx of sodium ions into the muscle cell. This initiates an action potential that propagates along the sarcolemma and into the T-tubules, triggering muscle contraction.
Acetylcholine binds to its receptor in the sarcolemma and triggers .?
Acetylcholine binds to its receptors on the sarcolemma, leading to the opening of ion channels that allow sodium ions (Na+) to enter the muscle cell. This influx of sodium ions depolarizes the sarcolemma, generating an action potential. The action potential then travels along the T-tubules, ultimately triggering the release of calcium ions from the sarcoplasmic reticulum, which initiates muscle contraction.
What best describes how ACh changes the ion permeability of the sarcolemma?
Acetylcholine (ACh) binds to nicotinic receptors on the sarcolemma of muscle fibers, leading to the opening of ion channels. This results in an influx of sodium ions (Na+) into the cell, which depolarizes the sarcolemma and initiates an action potential. The change in ion permeability is crucial for muscle contraction, as it propagates the signal to the interior of the muscle fiber and ultimately stimulates the release of calcium ions from the sarcoplasmic reticulum.
Which component has a role in the postsynapitc cell during synaptic activity?
chemically gated channels
Plasma membrane exhibits chemically gated ion channels?
Chemically gated ion channels in the plasma membrane are sensitive to specific molecules that bind to them, causing the channel to open or close. This allows for the controlled movement of ions across the membrane in response to chemical signals, regulating processes such as muscle contraction and neurotransmission.
When acetylcholine diffuses across the synaptic cleft and binds to receptors on the folded sarcolemma it initaly causes what channels to open in the sarcolemma?
Acetylcholine binding causes nicotinic acetylcholine receptors on the folded sarcolemma to open, allowing the influx of sodium ions into the muscle cell. This initiates an action potential that propagates along the sarcolemma and into the T-tubules, triggering muscle contraction.
What surrounds myofibrils?
The sarcoplasmic reticulum are the membranous interconnecting channels and sacs that surround and run parallel to the myofibrils.
Acetylcholine binds to its receptor in the sarcolemma and triggers .?
Acetylcholine binds to its receptors on the sarcolemma, leading to the opening of ion channels that allow sodium ions (Na+) to enter the muscle cell. This influx of sodium ions depolarizes the sarcolemma, generating an action potential. The action potential then travels along the T-tubules, ultimately triggering the release of calcium ions from the sarcoplasmic reticulum, which initiates muscle contraction.
What is the sarcolemma of a resting muscle fiber is most permeable to?
The sarcolemma of a resting muscle fiber is most permeable to potassium ions (K+). This permeability is primarily due to the presence of potassium leak channels, which allow K+ to flow out of the cell, contributing to the resting membrane potential. In contrast, the sarcolemma is less permeable to sodium ions (Na+), which helps maintain the negative charge inside the cell at rest.
What best describes how ACh changes the ion permeability of the sarcolemma?
Acetylcholine (ACh) binds to nicotinic receptors on the sarcolemma of muscle fibers, leading to the opening of ion channels. This results in an influx of sodium ions (Na+) into the cell, which depolarizes the sarcolemma and initiates an action potential. The change in ion permeability is crucial for muscle contraction, as it propagates the signal to the interior of the muscle fiber and ultimately stimulates the release of calcium ions from the sarcoplasmic reticulum.
Which component has a role in the postsynapitc cell during synaptic activity?
chemically gated channels
Which component has a role in the post synaptic cell during synaptic activity?
Chemically Gated Channels.
What causes depolarisation of sarcolemma of muscle fibre?
An incoming action potential to an alpha motor neuron causes acetylcholine (Ach)release at the end plate, Ach binds to Ach receptors on the sarcolemma causing a sodium influx which causes depolarization.
Plasma membrane exhibits chemically gated ion channels?
Chemically gated ion channels in the plasma membrane are sensitive to specific molecules that bind to them, causing the channel to open or close. This allows for the controlled movement of ions across the membrane in response to chemical signals, regulating processes such as muscle contraction and neurotransmission.
How does a nerve generates its action potential?
A nerve generates an action potential through a series of events involving the opening and closing of ion channels. Initially, a stimulus causes sodium channels to open, allowing an influx of sodium ions, depolarizing the cell membrane. This triggers the opening of voltage-gated sodium channels, leading to a rapid depolarization phase and the propagation of the action potential along the nerve.
Which membrane potential occurs because of the influx of Na plus through chemically gated channels in the receptive region of a neuron?
The membrane potential that occurs due to the influx of Na+ through chemically gated channels in the receptive region of a neuron is called the excitatory postsynaptic potential (EPSP). This influx of Na+ leads to depolarization of the neuron, bringing it closer to the threshold for generating an action potential. EPSPs can summate to trigger an action potential if they reach the threshold potential.
Do membranes of the dendrites contain chemically gated ion channels?
Yes, the membranes of dendrites contain chemically gated ion channels. These channels open or close in response to specific neurotransmitters binding to their receptors, allowing ions such as sodium, potassium, or calcium to flow into or out of the dendrite. This ion movement is crucial for generating electrical signals in dendrites and communication between neurons.
