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The resting membrane potential value for sodium is closer to the equilibrium of potassium because the sodium-potassium pump actively maintains a higher concentration of potassium inside the cell and a higher concentration of sodium outside the cell. This leads to a higher permeability of potassium ions at rest, resulting in the resting membrane potential being closer to the equilibrium potential of potassium.

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Through the membrane of a resting neuron highly permeable to potassium ions its membrane potential does not exactly match the equilibrium potential for potassium because the neuronal membrane is?

The neuronal membrane also has ion channels for other ions besides potassium, such as sodium or chloride, that can influence the resting membrane potential. These other ions contribute to the overall equilibrium potential of the neuron, which affects its resting membrane potential. Additionally, the activity of Na+/K+ pumps helps establish and maintain the resting membrane potential, contributing to the slight difference from the potassium equilibrium potential.


Why is there a resting membrane potential across the cell membrane?

sodium/potassium pump


The membrane-bound enzyme system that restores and maintains the resting membrane potential is what pump?

The membrane-bound enzyme system responsible for restoring and maintaining the resting membrane potential is the sodium-potassium pump. It actively transports sodium ions out of the cell and potassium ions into the cell against their concentration gradients to establish the resting membrane potential.


Ions used to establish a resting potential?

Potassium and sodium determine the a cell's resting membrane potential. The equilibrium potential (the voltage where no ion would flow) for sodium is about +60 mV while that for potassium is usually around -80 mV, but because the resting cell membrane is approximately 75 times more permeable to potassium than to sodium, the resting potential is closer the the equilibrium potential of potassium. This is because potassium leak channels are always open while sodium come in through voltage gated or ligand gated channels.


Explain why a change in extracellular sodium did not alter the membrane potential in the resting neuron?

A change in extracellular sodium concentration would not alter the resting membrane potential of a neuron because the resting potential is primarily determined by the relative concentrations of sodium and potassium ions inside and outside the cell, as mediated by the sodium-potassium pump and leak channels. Changes in extracellular sodium concentration would not directly affect this equilibrium.

Related Questions

Through the membrane of a resting neuron highly permeable to potassium ions its membrane potential does not exactly match the equilibrium potential for potassium because the neuronal membrane is?

The neuronal membrane also has ion channels for other ions besides potassium, such as sodium or chloride, that can influence the resting membrane potential. These other ions contribute to the overall equilibrium potential of the neuron, which affects its resting membrane potential. Additionally, the activity of Na+/K+ pumps helps establish and maintain the resting membrane potential, contributing to the slight difference from the potassium equilibrium potential.


What is the significance of the equilibrium potential in determining the resting membrane potential of a cell at cl equilibrium?

The equilibrium potential is important in determining the resting membrane potential of a cell because it represents the voltage at which there is no net movement of ions across the cell membrane. At this point, the concentration gradient and electrical gradient for a specific ion are balanced, resulting in a stable resting membrane potential.


Why is there a resting membrane potential across the cell membrane?

sodium/potassium pump


How does potassium affect the resting membrane potential of the cardiac cell?

Potassium plays a crucial role in maintaining the resting membrane potential of cardiac cells. It helps establish the negative charge inside the cell by moving out of the cell through potassium channels. This outward movement of potassium ions contributes to the polarization of the cell membrane, creating a negative resting membrane potential.


What is the significance of the cl- equilibrium potential in determining the resting membrane potential of a neuron?

The equilibrium potential for chloride ions (Cl-) plays a significant role in determining the resting membrane potential of a neuron. This is because the movement of chloride ions across the cell membrane can influence the overall balance of ions inside and outside the neuron, which in turn affects the resting membrane potential. If the equilibrium potential for chloride ions is altered, it can lead to changes in the resting membrane potential and impact the neuron's ability to transmit signals effectively.


The membrane-bound enzyme system that restores and maintains the resting membrane potential is what pump?

The membrane-bound enzyme system responsible for restoring and maintaining the resting membrane potential is the sodium-potassium pump. It actively transports sodium ions out of the cell and potassium ions into the cell against their concentration gradients to establish the resting membrane potential.


What are the forces at equilibrium potential?

At equilibrium potential, the forces on an ion are balanced, meaning there is no net movement of ions across the membrane. The electrical force due to the membrane potential balances the chemical force due to the concentration gradient, resulting in equilibrium. This can be seen in action for ions like potassium (K+) at its equilibrium potential in a resting neuron.


Ions used to establish a resting potential?

Potassium and sodium determine the a cell's resting membrane potential. The equilibrium potential (the voltage where no ion would flow) for sodium is about +60 mV while that for potassium is usually around -80 mV, but because the resting cell membrane is approximately 75 times more permeable to potassium than to sodium, the resting potential is closer the the equilibrium potential of potassium. This is because potassium leak channels are always open while sodium come in through voltage gated or ligand gated channels.


What effect did increasing the extracellular potassium have on the resting membrane potential?

Increasing the extracellular potassium concentration can depolarize the resting membrane potential, making it less negative. This can lead to increased excitability of the cell.


What does the potassium equilibrium potential of -94 mV signify in terms of cellular function and ion balance?

The potassium equilibrium potential of -94 mV indicates that the concentration of potassium ions inside and outside the cell is balanced. This balance is crucial for maintaining the cell's resting membrane potential and proper functioning of processes such as nerve signaling and muscle contraction.


Explain why a change in extracellular sodium did not alter the membrane potential in the resting neuron?

A change in extracellular sodium concentration would not alter the resting membrane potential of a neuron because the resting potential is primarily determined by the relative concentrations of sodium and potassium ions inside and outside the cell, as mediated by the sodium-potassium pump and leak channels. Changes in extracellular sodium concentration would not directly affect this equilibrium.


What ion determines the resting membrane potential of nerve and muscle?

The potassium ion (K+) plays a major role in determining the resting membrane potential of nerve and muscle cells. This is because these cells have a higher permeability to potassium ions than other ions, such as sodium ions. As a result, the movement of potassium ions out of the cell through potassium leak channels leads to the establishment and maintenance of the negative resting membrane potential.