Chicken is preferred to meat, but should still be limited two times in month . The chicken should not be cooked in fat and the skin should be removed.
Even though there is no specific cure available, there are effective medicines. They have to do with the thinning of the septum. They will not cure it completely, though it will relieve many symptoms (which itself can extend life).
Fentanyl Cardiovascular side effects:
Marijuana Cardiovascular side effects:
Heart disease is the leading cause of death worldwide. It is basically from our way of life, smoking, physical inactivity, stress. They are all related to heart disease.
I don't know but brain damage occurs after 3 minutes.
Heart failure means that your heart can no longer pump blood adequately through the heart and out to the rest of the body. The blood literally "backs up" into the pulmonary system (lungs) and your lungs become too fluid filled to exchange oxygen and carbon dioxide. This process deprives your body's tissues/organs from the oxygen it needs and they begin to fail. This can be a very slow process if it is a chronic condition, or it can occur very quickly depending on how quickly the heart function is failing, and why (example: heart failure due to chronic high blood pressure or heart attack which results in heart tissue death)
adj. unremarkable - 1. Lacking distinction; ordinary.2. found in the ordinary course of events; "a placid everyday scene"; "it was a routine day";
In medical practice, when nothing UNUSUAL found, occured or happened, its UNREMAKABLE
From Wiki s users...
I was diagnosed with viral cardiomyopathy in 2001. Leading up to the diagnosis I was getting more and more short of breath, sweating profusely, and some swelling. I was working night shift at the hospital (Respiratory therapist) and sitting at the table with a co-worker when she mentioned that I should go to the ER and get checked out, I did. The ECG came back reading Left Bundle Branch Block, We did an echocardiogram that AM and found I had an EF of 21%, (ejection fraction = The ejection fraction evaluates how well the heart is pumping; Normally 50 - 70 percent). 3 days later we did a heart cath and confirmed the findings from the echo. I was lucky that I found things early with no enlargement of my heart; I started taking Coreg 25mg BID and Altace 10mg QD no strenuous activity for the first year. In Dec 2006 on my last echo my EF is now 55% and I am taking much lower doses of the Coreg and Altace with the same results. I will be on these meds for life but I am still here and very thankful. My strongest recommendation is to listen to your cardiologist and DO WHAT THEY SAY. I was lucky s below a bit pessimistic. Cardiomyopathy and myocarditis are separate diseases. Viral myocarditis does recover if its not severe and you survive the acute illness (possibly with intensive care etc) It can also be treated by transplant if it is severe. Overall the mortality is said to be 50% and I lost a medical friend to it recently. Good luck. Unfortunately, it is a fatal disease. Patient have a survival expectancy of approx. five years after diagnosis. Antiarrhythmic and ACE Inhibitors are sometimes helpful in relieving some of the symptoms.
A glimmer of hope. My father was diagnosed with this disease in 1989 and survived until 2006. It began as pneumonia, progressed to double pneumonia and then the virus moved to his heart reducing the pumpage of the heart by 90%, that's right he had 10% of the normal person after the virus had run it's course. This man was disabled and would turn red and then purple walking less than 10 steps. He was forced into retirement at 45 (diesel mechanic) with 3 children still at home. He had too much to live for to die so he lost weight, followed all the doctors orders, and slowly worked his way back to 15% pumpage. He worked first on just walking around the house, then running errands, the more challenging things like tinkering with the lawn mower. Ten years after he was disabled the doctor released him to go back to very light duty mechanic (supervisory and teaching mostly)work part-time, later to increase it to full. He had a massive heart attack 15 years later that claimed his life, they had given him 6 months! During those fifteen years he saw his children graduate, marry, all of his grandchilren born and celebrated 40 years with his bride. The man even pastored a church again. He accomplished everything he wanted to before he passed. This time allowed me to know my Dad, so valuable. Never give up! The human spirit is a remarkable thing. If you've got faith in a higher power that helps too. You set the goals and beat the odds. In feb 2001, I was diagnosed with Viral cardiomyopathy, spent 2 months in a medically induced coma on heart/lung machine, and my heart "healed itself" while I was on the support system waiting for a transplant.
Today I'm back to my old 1 hour/day exerciseroutine, feel great and show no signs of the disease. I find the info such as the one you received earlier USELESS! I pushed myself to exercise ( I had to relearn to walk after so much bed rest), listening to my heart and resting when necessary, but DID NOT give up.
Too many people take it as a death sentence when in fact 50% of people recover according to my cardiologist! Go to a major institute, such as MD Andersen, Vanderbilt, etc. for treatment. It's worth your while and stay positive! The heart is a muscle and with training, it can be strengthened.
Hang in there and ignore the negatives! I was diagnosed with viral cardiomyopathy after suffering pancreatitis. My heart is very weak. I also had congestive heart failure in spite of having an artificial heart valve. I spent approximately 2 1/2 months in Temple Univ. Med. Ctr. I have been on a specific protocol and although I am weak at times, I do not feel that it is a death sentence. I'm 68 years old and hope to have another 5-10 years. I don't think many people can have a guarantee as to how long they can live. I have no blockages, kidney disease, pulmonary disease and if I didn't do well on the protocol I was in line for a heart transplant. Yes you do get quite tired, but you have to be aware that at those times you need to rest. The secret is not to overexert yourself. I was diagnosed with Viral Cardiomyopathy when I was 26. At the time I was working as a fitness instructor and going to graduate school. My diagnosis was not as severe as others subsequently none of my doctors said anything about dying in 5 years. I have not been as active as I once was but I am still living and have no plans of dieing soon. 7 years ago wife diag. with "VIRAL CARDIOMYOPATHY"-on medications since and seemly in good health--- NOT OVERWEIGHT--even now with ejection fraction at 15 she is quite active however she now will need pacemaker---The whole point is that since she was never overweight this in itself was a graet factor in her long term survial---no she does not exercise alot good luck youall I was diagnosed with viral cardiomyopathy when I was one and a half years old. I was also born with a Ventrical Septal Defect at birth and congestive heart failure. The doctors who delivered me said I wouldn't survive more than a few days. I was brought to Children's Heart Clinic under the care of Dr. Katkov in Minneapolis, Minnesota and he told my parents I would not die. I am now 20 years old and in school going for biology and genetics. I want to be a pediatriccardiologist. I won state for track three years in a row and I have been in dance since I was three. Now I coach danceline. So no, that is not true. As long as you believe and everyone around you believes, you will not die. When you have no hope for living, God sees that, and will do nothing. When you love life and enjoy every part of being on Earth, you will conquer. On Christmas Eve in 2004 I went to the emergency room knowing only that my heart was enlarged and that my regular doctor said I had pneumonia. After hours and hours of tests, they finally determined that I was having congestive heart failure and I was admitted. I was 32 years old. On Christmas day I had an eccocardiogram done and was told that my ejection fraction was 15%. I spent a whole week in the hospital while they tried to determine what had caused this to happen to me. I had no previous history of heart problems or any problems for that matter. They finally determined that the heart failure had been caused by viral pneumonia. I was released from the hospital and went back to work a week later. After 6 months I had another eccocardiogram and was told my ejection fraction was only 25%, but I felt fine. I was exercising every day and didn't really feel like there was anything wrong with me. I was told that the biggest improvement in your ejection fraction is made during the first 6 months. Because mine was still so low, they recommended an ICD. I had the ICD implanted within a couple of weeks. I have never been shocked, but just knowing it is there if I need it makes a world of difference. In June of this year I had another eccocardiogram done and was very pleased to find that my ejection fraction was now at 50%. Don't give up and don't think of yourself as a statistic. You have to decide that you are going not going to die. Viral cardiomyopathy is a rare thing, but you can recover from it.
As a rule-of-thumb, people with higher HDL have a lower incidence of cardiovascular disease (CVD). However, there's no definitive proof that HDL has a preventative effect on CVD. Higher HDL may be an indicator of some other process that reduces CVD. HDL is lowered by acute and chronic inflammatory states. For example, having a cold will temporarily lower HDL.
HDL does play a role in a process called reverse cholesterol transport. Whether that process reduces CVD is not certain. HDL also transports anti-oxidant enzymes throughout the body, which may explain some of its beneficial effects.
There have been few studies demonstrating that raising HDL reduces CVD. For example, niacin has been shown to reduce CVD mortality, although it's not proven that it's because it raises HDL. Most studies have only increased HDL by 10%, which may not be enough for a significant clinical effect.
Diet does play a role in HDL levels. Diets that are high in refined carbs or low in fat reduce HDL. On the other hand, low carb diets high in saturated fats can profoundly increase HDL levels. On of the most effective saturated fatty acids that increases HDL is lauric acid, which is primarily found in coconut.
Recent studies have also shown that saturated fat consumption is not associated with CVD. The majority of studies that claim to show a link failed to control for other factors such as sugar or polyunsaturated fat consumption.
A cardiologist (cardi/o meaning heart and -ologist meaning specialist) treats diseases of the heart and blood vessels. If surgery is required you may need to see a thoracic surgeon.
what is the medical term meaning obtaining fluid from a joint in order to examine for diagnosis
the heart stops beating and death normally occurs right away
Vitamin E is a generic term for tocopherols and tocotrienols. Vitamin E is a family of α-, β-, γ-, and δ- (respectively: alpha, beta, gamma, and delta) tocopherols and corresponding four tocotrienols. Vitamin E is a fat-soluble antioxidant that stops the production of reactive oxygen species formed when fat undergoes oxidation. Of these, α-tocopherol (also written as alpha-tocopherol) has been most studied as it has the highest bioavailability.Congenital heart defectsA case control study done in the Netherlands using food frequency questionnaires found that high maternal Vitamin E by diet and supplements is associated with an increased risk of CHD (congenital heart defects) offspring, especially when the supplements are taken in the periconception period. (Note: case control studies are rated as low quality, grade 3 or 4, on a standard scale of medical evidence.) The National Health Service in the United Kingdom concludes that pregnant women should: "consider avoiding taking supplemental Vitamin E tablets." Vitamin E and strokesA Finnish study found that Vitamin E supplementation increased the risk of hemorrhagic stroke. Vitamin E supplementation was shown to increase the risk of heart failure in a 2005 study.
Heart disease is a general term which describes many malfunctions of the heart. Most of us envision the "hardening of the arteries" type of illness, but heart disease encompasses electrical problems within the heart, weakening of the heart muscle, congenital defects, valve malfunctions, and many other issues. Each type of illness brings a unique treatment and affects people differently.
The heart is responsible for bringing blood and oxygen to the brain and body, resulting in decreased brain function, body strength and stamina. Since some types of heart disease can develop over time, causing many people attribute these changes to aging rather than illness.
Some heart disease, such as Sudden Cardiac Arrest, occurs quickly and can cause death.
It is important to discuss your risk of heart disease, including family history, with your doctor. Your risk can be reduced, and the affect on your life limited, by eating well, moving more, and managing stress.
Date of Birth: 05/03/1903 Date of Death:10/14/1977 Age at Death: 74
Cause of Death:
Heart attack Go to --> www.deadoralive.com <---- to find out about famous people. Just type in a name.
had pain in right lung,had xray and anti biotics amoxicillan,pain gone but lung feels slightly gravely
The fatty deposits of cholesterol within the artery wall are known as atherosclerotic plaque or atheromatous plaque. It is divided into three different components -
1) The atheroma, the nodular accumulation of soft, flaky, yellowish material, composed primarily of spent macrophages nearest the lumen of the artery
2) Underlying areas of cholesterol crystals
3) Calcification at the outer base of older/more advanced lesions.
The 'atheroma', as it is also known, which is comprised of cells (mainly macrophage cells), cell debris, lipids (cholesterol and fatty acids), calcium and a variable amount of fibrous connective tissue.
If/when one of these plaques ruptures, to due structural collapse, it causes a clot in the artery, leading to a heart attack, cardiac arrest, or stroke.
Atherosclerosis is a condition in which an artery wall thickens as a result of the buildup of fatty materials. Atherosclerosis results in a reduced flow of blood in the affected artery. Atherosclerosis can develop in the coronary arteries, which supply blood to the heat muscle.
Depending on what caused the congestive heart failure. Normally diuretics such as caffeine are dangerous as they can lead to cardiac arrest for weak hearts.
Angioplasty is a popular method of surgery for curing blocks in the heart arteries due to plaque buildup. In this method a balloon catheter is injected into a narrow artery & later inflated to broaden the artery. Hence this method is also called balloon angioplasty.
I am very sorry to hear about your beloved pet and I know it's not easy to see them ill. I've had to go through this several times myself with many of my past pets. Congestive heart failure is biding for time with your pet. The vet is doing everything he/she can. It's time you went and saw your vet and simply told him/her that when they think your pet is no longer enjoying life and their health problems are causing them stress to let you know when to put your pet to sleep. I know this is heart breaking, but the pet owner must have empathy as to how much their pet is suffering. Dogs are much like people and when they have Congestive heart failure their appetite can go down a great deal and also it could be the signs of your pet's heart simply giving out. When the heart is this bad it affects other organs and it's simply a matter of time. Generally the heart is surrounded by fluid and the dog may go into pneumonia. I had a Cairn Terrier (the love of my life) and she had Cushing's Disease (cancer.) I had the lump removed, but she was never quite the same after that. Our vet suggested cancer treatment, but I read enough about it that I decided not to go down this road (not because of money issues) but the quality of our beloved pet's few good years left. We discussed this with our vet and my husband and I slowed down our pace of life and spent quality of time with our Daisy. We had wonderful times together and every couple of weeks I'd take her in mainly to be sure she was not in pain. She never was and she became very ill 10 months later (on a long weekend of course) and we had to rush her off to ER. We stayed with her for hours and it was evident that she wouldn't be with us much longer and we were there when she was put to sleep. We were heart-broken, but thankful she didn't have to suffer any longer. Please talk candidly to your vet and go from there.
my sister was just been told this is her problem as they have delayed her surgey on her colon how serious is this
Fibrillation is the medical term for what your heart does when it is not beating correctly. Defibrillator is the devise used to send a electric shock to your heart to stimultate it back into its regular beating mode, ---------------------------------------------------------------------------------------------------------------
Normally the heart cells fire in a orderly way controlled by natural pacemaker sites that are part of the heart. When the heart is fibrillating, cells are not firing in an organized way, the pacemaker sites have lost control. Often there is no damage YET and a heart attack, ( by definition causes an area of damage to the heart), has not happened yet.
If just the top of the heart is fibrillating the defibrillation ( in this case often called a cardioversion) can sometimes be delayed without damage and the patient will remain conscious. If the bottom part or the whole heart is fibrillating they will not have a pulse, will often have a convulsion and will stop breathing. Defibrillation in this case must be done immediatly. If done within one minute there is up to a 90% chance of survival dropping 10% for each minute the defibrillation is delayed. There is no way that any EMS system can get there fast enough, that is why we need AEDs in areas of public assembly or high risk areas like Health Clubs and schools.
Defibrillation resets the heart with an electrical discharge that fires all the muscle cells in the body ( that is why the body jerks when you push the button to fire the defibrillator. This resets the heart, like when you reset your computer. For a moment it actually stops all activity in the heart. If you were looking at a heart monitor you would see the activity go to flat line ( asystole) then, once ANY one of the three pacemaker sites cature the rhythm the heart can beat normally.
If the fibrillation has caused sudden cardiac arrest the defibrilation must be done within 3-5 minutes from the time the heart fibrillates or the chances for survival drop below 50%. The average response time at BEST in the US is 4-10 minutes. The chance of survival drops 7-10% per minute.
In the best paramedic systems in the US survival of out of hospital cardiac arrest is 2%-15%. In locations that have Automated External Defibrillators that have literally been used by 6th graders with minimal training and can NOT cause harm the survival rate improves to 70% or better.
A difibrillator is the device that doctors, or regular people, use to start one's heart up again. It is the machine that shocks the heart so it beats again.
what does a defibrillator look like
A piece of equipment used in resussitation when someone is in cardiac arrest or other cardiac related life threatening problems. It delivers a theraputic amount of electrical energy to the affected area of the heart to allow normal sinus rhythm.
Ischemia means a reduced blood supply to an organ or body part.
Antero- refers to the anterior, or front side of something.
-lateral means to the side or the outside of something.
So to put it all together Anterolateral Ischemiameans that there is or was a reduction of blood flow to the front outside part (of an organ).
This is often a term used to describe findings after chest pain or a heart attack. In that case it would mean that the area of the heart on the front side and just to the outside (either right or left) of a line drawn down the middle of the heart has had a reduction in blood supply with some damage to the heart muscle tissue (either temporary or permanent).
The heart muscle is called Myocardial tissue.
Myo- is muscle
cardio- refers to heart.
So, if this is describing reduction in blood supply to the outside front part of the heart muscle... that would be called Anterolateral Myocardial Ischemia.
A defibrillator is primarily used for two conditions, Ventricular Fibrillation (V-Fib) and Ventricular Tachycardia (V-Tach). V-Fib is when the heart basically twitches and V-Tach is when it beats too fast to actually move blood. A defibrillator will NOT work if there is no electrical rhythm. If the heart is not beating and has no electrical activity at all (Asystole) then contrary to what happens in the movies, using a defibrillator would be pointless. In this situation, the only thing that can possibly "bring the patient back" is full life-support and cardiac drugs.
Few opportunities to "bring back" one who has died are as time sensitive as defibrillation. With trauma we have the golden hour. In the best EMS systems in the US with the fastest response times the save rate for out of hospital cardiac arrest is only 2%-15%. We are making some major changes for the better the last 2 years with the advent of Public Access Defibrillators, Hands Only CPR, and cerebral resusitation such as rapid cooling and induced comas in the hospital.
We need to get a patient in Sudden Cardiac Arrest defibrillated in 1 to 3 minutes for a 70%-90% resusitation rate. Waiting the 8 minutes average response time for first responders equates to a 20% save rate or less.
Any time a patient is not breathing and appears to be dead ( with few exceptions that it would not be appropriate to go into here) you should apply the AED as soon as possible. Do not delay CPR to wait for the AED but immediatly on arrival of the AED stop CPR as the pads are ready to be applied. If the arrest is witnessed ( if they have been down less then 5 minutes) immediatly allow the AED to analize the rhythm and follow it's instructions. If there will be a delay in arrival of the AED begin rapid compressions without breathing stopping when the AED has arrived and the pads are ready to be placed. If you have oxygen available put them on high flow oxygen with a nonrebreathing mask, air will be moved as in breathing as you compress and allow the chest to expand, after all, that is how you are breathing as you read this.
If you do not know how long the patient has been down but it is likely more then 5 minutes do 200 compressions prior to applying the AED then follow it's instructions.
If you are not currently certified in AED but have been trained in the past most States include you in the Good Samaritan limited immunity statutes. If nobody is using the AED then you should use it. The machine is so smart and so fail-safe that 6th graders with minimal training have demonstrated they can effectivly and safely use it.
An AED can not shock a patient who is breathing and is not in a cardiac rhythm that requires a rapid shock to survive. No matter what you do or how many times you push the button you CAN NOT shock a patient who's life does not depend on you doing so.
You do NOT have time to wait for Fire/EMS/PD to arrive, if you see or know there is an AED there and the patient is not breathing turn it on, follow the instructions given regardless of if your training is current or not.
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