Out of the cell
As the action potential passes an area on the axon, sodium channels are closed, preventing influx of more sodium ions. At the same time, voltage-sensitive potassium channels open, allowing the membrane potential to fall quickly. After this repolarization phase, membrane permeability to potassium remains high, allowing for the "afterhyperpolarization" phase. During this entire period, while the sodium ion channels are forced closed, another action potential cannot be generated except by a much larger input signal. This helps to prevent the action potential from moving backwards along the axon.
exocitocisThey dir
A traveling action potential passes channels that allow ions to flow in both directions across the membrane, which very rapidly restores the potential of the membrane traversed. In the most simple configuration, influx of sodium (Na+) is quickly balanced by efflux of potassium (K+).Beware confusing this with how a cell's initial ion gradients are restored (e.g. sodium potassium pumps in the membrane), or occasional directionally-biased placement of ion-specific channels (e.g. nodes of ranvier), or channel reactivation (molecular voltage gates in channels).
When a nerve impulse is conducted, the neuronal cell membrane undergoes changes in electrical potential. This starts with a rapid influx of sodium ions into the cell through voltage-gated sodium channels, depolarizing the membrane. This depolarization triggers the opening of adjacent sodium channels, resulting in an action potential that travels along the membrane. After the impulse passes, the sodium channels close, and potassium channels open, allowing potassium ions to exit the cell and restore the resting potential.
Acetylcholine (ACh) is the neurotransmitter that passes from neuron to muscle cells and it causes the Na/K channels to open allowing Na+ to flow into the cell triggering an action potential.
As the action potential passes an area on the axon, sodium channels are closed, preventing influx of more sodium ions. At the same time, voltage-sensitive potassium channels open, allowing the membrane potential to fall quickly. After this repolarization phase, membrane permeability to potassium remains high, allowing for the "afterhyperpolarization" phase. During this entire period, while the sodium ion channels are forced closed, another action potential cannot be generated except by a much larger input signal. This helps to prevent the action potential from moving backwards along the axon.
As the action potential passes an area on the axon, sodium channels are closed, preventing influx of more sodium ions. At the same time, voltage-sensitive potassium channels open, allowing the membrane potential to fall quickly. After this repolarization phase, membrane permeability to potassium remains high, allowing for the "afterhyperpolarization" phase. During this entire period, while the sodium ion channels are forced closed, another action potential cannot be generated except by a much larger input signal. This helps to prevent the action potential from moving backwards along the axon.
I believe that you are referring to a "reflex".
exocitocisThey dir
A traveling action potential passes channels that allow ions to flow in both directions across the membrane, which very rapidly restores the potential of the membrane traversed. In the most simple configuration, influx of sodium (Na+) is quickly balanced by efflux of potassium (K+).Beware confusing this with how a cell's initial ion gradients are restored (e.g. sodium potassium pumps in the membrane), or occasional directionally-biased placement of ion-specific channels (e.g. nodes of ranvier), or channel reactivation (molecular voltage gates in channels).
When a nerve impulse is conducted, the neuronal cell membrane undergoes changes in electrical potential. This starts with a rapid influx of sodium ions into the cell through voltage-gated sodium channels, depolarizing the membrane. This depolarization triggers the opening of adjacent sodium channels, resulting in an action potential that travels along the membrane. After the impulse passes, the sodium channels close, and potassium channels open, allowing potassium ions to exit the cell and restore the resting potential.
Acetylcholine (ACh) is the neurotransmitter that passes from neuron to muscle cells and it causes the Na/K channels to open allowing Na+ to flow into the cell triggering an action potential.
Potential difference is defined as follows: every coloumb of charge that passes through this difference will gain (or lose, depending on direction and signs) 1 joule of energy. This unit, joule/coloumb, is simply called the volt.
potential energy changes to kinetic energy
When light changes direction as it passes through a boundary.
The direction of the wave will eventually change to where the wind is blowing.
After a neuron fires, there is a refractory period where certain factors in the neuron prevent it from being depolarized again. This is made possible by an "overshoot" of polarization (returning to a much stronger negative ion charge inside the neuron) after the action potential passes. This ensures that voltage-controlled ion channels remain closed for a small period of time and do not become overactive through continuous restimulation.