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When your nose is stuffy cannot reach nerve receptors to initiate an impulse that would result in smell or taste?
Wiki User
∙ 14y ago
chemicals
Wiki User
∙ 14y ago
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What does spontaneous improvisation mean?
It was literally mean occuring as a result of unplanned impulse.
Which one of newton's laws is a direct result of impulse-momentum relationship?
yes
Why does a reaction force exist in a airplane?
Forcing backwards will result on a impulse forwards.
Are pain receptors slow or fast adapting receptors?
Pain receptors, also known as nociceptors, are primarily slow-adapting receptors. This means that they continue to send signals for an extended period of time in response to a painful stimulus. They are designed to detect potential threat or damage to the body and provide a persistent awareness of pain.
What is the dynamic tension that demands onward motion in music usually the result of?
Dynamic tension that demands onward motion in music is usually the result of the impulse of dissonance to be resolved.
What would result if your olfactory receptors were damaged?
Your sense of smell will be lost or diminished.
What parts of brain do the neurolyptics effect?
The theory behind why individuals develop 'psychotic' symptoms is based upon the idea that there are elevated levels of dopamine in the brain. Dopamine is a neurotransmitter, a molecule that passes messages between neurons. For example, when a nerve impulse arrives at a dopaminergic neuron (also known as a pre-synaptic neuron), dopamine is released from the cell and diffuses through a space between two neurons, called the synaptic cleft. Dopamine then binds to specific dopamine receptors on a different neuron (post-synaptic neuron) producing a specific signal, impulse or effect. Dopamine is then released from its receptors and 're-absorbed' into the pre-synaptic neuron, or degraded by enzymes in the synaptic cleft. The neuroleptics block dopamine receptors thereby inhibiting the ability of dopamine to attach to these receptors and generate signals. However, unlike the typical neuroleptics, the atypicals merely transiently block the receptors therefore allowing some dopamine to bind to the receptors and generate signals. The atypical neuroleptics are also able to block serotonin receptors located on dopaminergic neurons. When serotonin binds to these receptors it inhibits dopamine release. However as these receptors are blocked by atypical neuroleptics, the dopamine secretion is increased. The transient rather than permanent blocking of dopamine receptors and the blocking of serotonin receptors and subsequent increases in dopamine, it is for these reasons that the atypicals are thought to produce fewer adverse effects than the typical neuroleptics. However, the atypical drugs differ in their 'stickyess' when binding to dopamine receptors and also in the ratio of which dopamine ad serotonin receptors are affected. This may result in some atypicals producing higher levels of specific adverse effects than others. The atypicals may also bind to other receptor types, producing further adverse effects (see side effects of atypicals section).
How many receptors does the blind spot have?
The "blind spot" is the point at which the optic nerve leaves the eye. Think of it as a cable attached to the back of the eye, carrying all your visual information to the brain. As a result, there are no receptors at the "blind spot".
What is the similarity of G-protein receptor system and Tyrosine-kinase receptor system?
both are plasma membrane receptors located at the cell surface's membrane. both are proteins that are hydrophilic and therefore cannot cross the lipid bilayer and requires second messengers to communicate with the nucleus of cells. activation of the receptors will activate other molecules in the activating cascades and the numbers of affected molecules will increase geometrically. however, activation of such receptors will cause desensitization/adaptation of the receptors themselves. the disability to desensitize can result to serious diseases such as cancer. Khairul Abu Bakar 2nd Year Medical Student National Univ.of Ireland, Galway
What physically causes the trip on LSD?
As simply stated as possible: Lysergic acid diethylamide (LSD) bonds to seven-transmembrane domain receptors associated with nerotranmitters. It bonds easily with all dopamine receptors and subtypes, all adrenoreceptor subtypes, and most serotonin receptors. Activating these receptors in neurotransmitters results in euphoria, the physiological manifestations of LSD use, and can (often does) result in aural, visual, and somatic (kinetic) hallucinations.
Touch is the result of receptors on the skin that can detect all except?
A+ i know the answer and u dont:) look it up somewhere else its not here -tl
What is the difference between feel and sense?
There are many sense organs in the body like touch receptors, pressure receptors, heart receptors, taste receptors, sound receptors, etc. All these receptors when stimulated result in changes in tone or isometric contraction of skeletal muscles around them or in the entire body.Sensation:Sensation is the observation of change in tone of skeletal muscles induced by a single sense organ.Thus, heat, sound, etc are sensations.Feeling:Feeling is the observation of changes in tone of skeletal muscles induced by many sense organs or continuous changes in a single sense organ.Thus, coffee induces a feeling and so does a song.
