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Resting Potential
The event in which a neuron's membrane potential rapidly rises from its resting potential and then falls back to its resting potential is called an action potential.The neuron fires an action potential and returns to its resting state in the following manner:Initially the resting potential of the inside of the cell membrane of a neuron with respect to the outside is about -70mV (this condition is referred to as polarized).As neural signals from inputs at the dendrites of the neuron move down the dendrites and across the soma (cell body), they arrive at the beginning of the axon, called the axon hillock; those signals are comprised of quantities of sodium ions which have been pushed to the axon hillock by an influx of sodium ions through ligand-gated sodium ion channels (ion pores which open from the action of a chemical messenger neurotransmitters in a receptor portion of the ion gate) in the dendrites which have been opened by neurotransmitters released by a pre-synaptic neuron diffusing across the synaptic cleft into receptors at the dendrite.Firing: If enough quantity of sodium ions reach the axon hillock to raise the membrane potential at that point to a threshold value of about -55mV(the trigger voltage), this is sufficient to open voltage-gated sodium ion pores in the initial segment of the axon, which allows more sodium ions in, raising the membrane voltage to from 50mV to 100mV (called depolarization), which cause nearby v-gated Na ion pores to open, which lets in more sodium ions, which open successive v-gated ion pores along the length of the axon. This moving (action) potential (voltage) is the neural impulse.Returning to resting state: during the peak of the action potential, when the membrane potential is at it greatest, sodium pores begin to close, and potassium pores are opened, and since there is more potassium inside the cell than outside, potassium ions begin to leave the neuron through those channels; with the loss of these positively charged ions, the membrane voltage becomes more and more negative, opening more potassium pores, until the membrane voltage actually undershoots the resting potential momentarily. At this point the potassium pores begin to close, and the membrane potential rises back to the resting potential.(please see the links below for additional explanations)
A nerve fiber becomes polarized when the resting potential of the membrane changes. It starts out with an unequal distribution of charges- the outside is more positive and the inside is less positive. (Sodium (Na+) is in a higher concentration on the outside of the membrane and Potassium (K+) is in a lower concentration on the inside of the membrane.) A stimulus changes the gradient- when more Na+ flows in, the resting potential changes and polarization occurs, allowing for an action potential to be propagated down the axon.
A neuron in its resting state, or resting potential, is not conducting an action potential, so its outside it is positive. It is only when it is conducting an action potential that it becomes depolarized and changes so its outside is negatively charged. The interior of a neuron's axon is negatively charged due to the presence of proteins and chloride ions both bearing negative charges. The chloride ions ions are able to pass through the cell membrane, although I do not recall if that movement is exclusively through voltage-gated channels.
The resting potential is the normal equilibrium charge difference (potential gradient) across the neuronal membrane, created by the imbalance in sodium, potassium, and chloride ions inside and outside the neuron.
Resting Potential
The event in which a neuron's membrane potential rapidly rises from its resting potential and then falls back to its resting potential is called an action potential.The neuron fires an action potential and returns to its resting state in the following manner:Initially the resting potential of the inside of the cell membrane of a neuron with respect to the outside is about -70mV (this condition is referred to as polarized).As neural signals from inputs at the dendrites of the neuron move down the dendrites and across the soma (cell body), they arrive at the beginning of the axon, called the axon hillock; those signals are comprised of quantities of sodium ions which have been pushed to the axon hillock by an influx of sodium ions through ligand-gated sodium ion channels (ion pores which open from the action of a chemical messenger neurotransmitters in a receptor portion of the ion gate) in the dendrites which have been opened by neurotransmitters released by a pre-synaptic neuron diffusing across the synaptic cleft into receptors at the dendrite.Firing: If enough quantity of sodium ions reach the axon hillock to raise the membrane potential at that point to a threshold value of about -55mV(the trigger voltage), this is sufficient to open voltage-gated sodium ion pores in the initial segment of the axon, which allows more sodium ions in, raising the membrane voltage to from 50mV to 100mV (called depolarization), which cause nearby v-gated Na ion pores to open, which lets in more sodium ions, which open successive v-gated ion pores along the length of the axon. This moving (action) potential (voltage) is the neural impulse.Returning to resting state: during the peak of the action potential, when the membrane potential is at it greatest, sodium pores begin to close, and potassium pores are opened, and since there is more potassium inside the cell than outside, potassium ions begin to leave the neuron through those channels; with the loss of these positively charged ions, the membrane voltage becomes more and more negative, opening more potassium pores, until the membrane voltage actually undershoots the resting potential momentarily. At this point the potassium pores begin to close, and the membrane potential rises back to the resting potential.(please see the links below for additional explanations)
A nerve fiber becomes polarized when the resting potential of the membrane changes. It starts out with an unequal distribution of charges- the outside is more positive and the inside is less positive. (Sodium (Na+) is in a higher concentration on the outside of the membrane and Potassium (K+) is in a lower concentration on the inside of the membrane.) A stimulus changes the gradient- when more Na+ flows in, the resting potential changes and polarization occurs, allowing for an action potential to be propagated down the axon.
Action potential is the term for an electrical change in the neuronal membrane transmitted along an axon. The axon is part of a nerve cell that conducts impulses.
The type of potential described is an action potential. It is generated by the movement of ions such as sodium and potassium across the axon membrane, leading to a rapid change in voltage that allows for the transmission of signals along the neuron.
A neuron in its resting state, or resting potential, is not conducting an action potential, so its outside it is positive. It is only when it is conducting an action potential that it becomes depolarized and changes so its outside is negatively charged. The interior of a neuron's axon is negatively charged due to the presence of proteins and chloride ions both bearing negative charges. The chloride ions ions are able to pass through the cell membrane, although I do not recall if that movement is exclusively through voltage-gated channels.
inward movement of sodium will increase and the membrane will depolarize.
ions
The resting potential is the normal equilibrium charge difference (potential gradient) across the neuronal membrane, created by the imbalance in sodium, potassium, and chloride ions inside and outside the neuron.
Inward movement of sodium ions will increase and the membrane will depolarize
I belive the size of the axon potential remains constant at a depolarisation of +40 mv and a resting potential of -70mv for most nerves. The frenquency of action potentials is the factor that determines the strength of the nerve impulse.
It causes the vesicles (which are in the axon terminal) to move to the cell membrane at the end of the axon terminal, where they merge with the cell membrane, releasing their load of neurotransmitters into the synaptic cleft (gap), where they quickly diffuse to receptors in the post-synapticneuron's dendrites, initiating a graded potential which moves down the dendrites, along the soma,to the axon hillock where it can cause an action potential in that secondneuron.