It causes the vesicles (which are in the axon terminal) to move to the cell membrane at the end of the axon terminal, where they merge with the cell membrane, releasing their load of neurotransmitters into the synaptic cleft (gap), where they quickly diffuse to receptors in the post-synaptic
neuron's dendrites, initiating a graded potential which moves down the dendrites, along the soma,
to the axon hillock where it can cause an action potential in that second
neuron.
Ether blocked the impulse transmission.
It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,and it can mask digitalis toxicity.
Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
When a stimulus stimulates a neuron above the threshold, the action potential is generated.
It creates an action potential
Depends on disease
Ether blocked the impulse transmission.
what effect does the drug quabain have on neuron
It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,and it can mask digitalis toxicity.
A synapse and an action potential have a flip-flopping cause and effect relationship, in that an action potential in a presynaptic neuron initiates a release of neurotransmitters across a synapse, which can then subsequently potentially trigger an action potential in the axon of the postsynaptic neuron, which would then cause release of neurotransmitters across a following synapse.
Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
They both decrease action potential duration, but TTX is the only one that decreases the maximum rate of depolarization.
It makes the muscle totally relax as it blocks the action potential in the nerves.
It blocks the sodium channels that are required to create action potential in the muscles to make them contract.
A negative effect is any result of an action which is perceived to be detrimental. This means its a subjective opinion. What might be negative to you may be positive to someone else.