G6PD deficiency is caused by one copy of a defective G6PD gene in males or two copies of a defective G6PD gene in females. Hemolytic anemic attacks can be caused by oxidants, infection, and or by eating fava beans.
Glucose-6-phosphate dehydrogenase deficiency is an inherited condition caused by a defect or defects in the gene that codes for the enzyme, glucose-6-phosphate dehydrogenase (G6PD).
The hematocrit test for the red blood cell content in blood for iron deficiency, the visual examination of the neck for iodine deficiency, or the examination of bones by densitometry for calcium deficiency.
sodium does not have a deficiency of electrons but has an extra electron in its outermost shell,which it loses to form an ionic bonds.
iodine
The conversion of glyceraldehyde 3 phosphate to 1, 3 bisphosphoglycerate catalyzed byglyceraldehyde 3 phosphate dehydrogenase using NAD+ and Pi
What disease can i get through lack of sulphur
No. They won't accept your blood
Glucose 6 phosphate dehydrogenase is an enzyme necessary for the metabolism of red blood cells. Its deficiency leads to hemolytic anemia usually triggered by exposure to some drugs.
medium-chain acyl-CoA dehydrogenase, MCAD, is a deficiency of enzymes that break down medium-chain fats such as coconut oils.
Most commonly this is from a deficiency of the enzyme that degrades alcohol (alcohol dehydrogenase). This is very common in Asians.
ADH (alcohol dehydrogenase) ADH (alcohol dehydrogenase)
alpha-ketoglutarate dehydrogenase is most similar to pyruvate dehydrogenase as both are enzyme complexes made of 3 units
Many Asians tend to have a deficiency in Acetaldehyde Dehydrogenase which breaks down the acetaldehyde that can accumulate in the brain after consumption of alcohol. This is what gives you the "hang over" affect.
Pyruvate dehydrogenase complex react with pyruvate to form acetyl-CoA
Lactate dehydrogenase is better known as lactic acid dehydrogenase. It is also abbreviated as LDH. If there is anything else it is called you can search for it on a medical website.
dehydrogenase is an enzyme which transfers hydrogen ions from a substrate to an activator.Example are activators are NAD+ and FAD+.
Provided the child is more than a few months old, does not have glucose-6-phosphate dehydrogenase deficiency (G6PD), and is not allergic to sulfonamides - then yes, Septra is generally a safe drug for children.
Malonic acid is a competitive inhibitor of succinate dehydrogenase.