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If a person gets methemoglobinemia from a certain medication that medication should be avoided at all costs in the future. For people with congenital methemoglobinemia medications or other things that are known to oxidize hemoglobin should be avoided.

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What is the prognosis for congenital methemoglobinemia?

Congenital methemoglobinemia is typically benign and should be observed. If methemoglobinemia symptoms occur the person should be taken to the hospital for treatment.


What is the prognosis for acquired methemoglobinemia?

If found early, acquired methemoglobinemia can be easily treated with no side effects. After treatment with methylene blue the patient can expect a full recovery.


How is acquired methemoglobinemia treated?

For acquired methemoglobinemia the typical treatment is with methylene blue. This is administered with an IV over a five-minute period and results are typically seen within 20 minutes.


What is methanoglobnemia?

Methanoglobnemia is mispelled, it's actually methemoglobinemia Methemoglobinemia is a blood disorder in which an abnormal amount of methemoglobin -- a form of hemoglobin -- is produced. Hemoglobin is the molecule in red blood cells that distributes oxygen to the body. Methemoglobin cannot release oxygen. In methemoglobinemia, the hemoglobin is unable to release oxygen effectively to body tissues.


Is methemoglobinemia an example of an x linked disorder?

Methemoglobinemia is not primarily classified as an X-linked disorder; it is usually caused by genetic mutations that affect the hemoglobin's ability to carry oxygen. The most common forms of hereditary methemoglobinemia are autosomal recessive, linked to mutations in the cytochrome b5 reductase gene. However, certain acquired forms can occur due to exposure to specific chemicals or drugs. While some genetic conditions are X-linked, methemoglobinemia does not fall into that category.


When was Methemoglobinemia discovered?

Methemoglobinemia was first described in 1943 by a physicians Alfred Stengel and Albert Mason of the University of Pennsylvania. They identified it as a condition where hemoglobin is unable to carry oxygen effectively due to the presence of excess methemoglobin in the blood.


What causes acquired methemoglobinemia?

Acquired methemoglobinemia can be caused by exposure to certain medications, chemicals, or substances that can oxidize hemoglobin, converting it to methemoglobin. Common causes include nitrate or nitrite medications, certain anesthetics, certain antibiotic drugs (such as dapsone and sulfonamides), and certain chemicals used in industries like aniline dyes or certain pesticides.


The Fugate family of Kentucky intermarried and passed on the recessive gene behind a disease called methemoglobinemia which causes an enzyme deficiency that results in cyanosis As a result the Fuga?

Yes, the Fugate family of Kentucky, also known as the "Blue Fugates," intermarried which concentrated the recessive gene causing methemoglobinemia. Methemoglobinemia is a condition where the blood is unable to carry sufficient oxygen due to the enzyme deficiency, resulting in a blue skin discoloration known as cyanosis in affected individuals of the family. This unique genetic trait in the Fugate family gained attention from medical researchers due to its rare and isolated occurrence.


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